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Record W2905141927 · doi:10.1158/0008-5472.can-18-2726

Genetic Data from Nearly 63,000 Women of European Descent Predicts DNA Methylation Biomarkers and Epithelial Ovarian Cancer Risk

2018· article· en· W2905141927 on OpenAlexafffund
Yaohua Yang, Lang Wu, Xiang Shu, Yingchang Lu, Xiao-Ou Shu, Qiuyin Cai, Alicia Beeghly‐Fadiel, Bingshan Li, Fei Ye, Andrew Berchuck, Hoda Anton‐Culver, Susana Banerjee, Javier Benı́tez, Line Bjørge, James D. Brenton, Ralf Bützow, Ian Campbell, Jenny Chang‐Claude, Kexin Chen, Linda S. Cook, Daniel W. Cramer, Anna DeFazio, Joe Dennis, Jennifer A. Doherty, Thilo Dörk, Digna Velez Edwards, Peter A. Fasching, Renée T. Fortner, Simon A. Gayther, Graham G. Giles, Rosalind Glasspool, Ellen L. Goode, Marc T. Goodman, Jacek Gronwald, Holly R. Harris, Florian Heitz, Michelle A.T. Hildebrandt, Estrid Høgdall, Claus Høgdall, David G. Huntsman, Siddhartha Kar, Beth Y. Karlan, Linda E. Kelemen, Lambertus A. Kiemeney, Susanne K. Kjær, Anita Koushik, Diether Lambrechts, Nhu D. Le, Douglas A. Levine, Leon F.A.G. Massuger, Keitaro Matsuo, Taymaa May, Iain A. McNeish, Usha Menon, Francesmary Modugno, Álvaro N.A. Monteiro, Patricia G. Moorman, Kirsten B. Moysich, Roberta B. Ness, Heli Nevanlinna, Håkan Olsson, N. Charlotte Onland‐Moret, Sue K. Park, James Paul, Celeste Leigh Pearce, Tanja Pejović, Catherine M. Phelan, Malcolm C. Pike, Susan J. Ramus, Elio Ríboli, Cristina Rodríguez‐Antona, Isabelle Romieu, Dale P. Sandler, Joellen M. Schildkraut, Veronica Wendy Setiawan, Nadeem Siddiqui, Weiva Sieh, Meir J. Stampfer, Rebecca Sutphen, Anthony J. Swerdlow, Lukasz M. Szafron, Soo‐Hwang Teo, Shelley S. Tworoger, Jonathan P. Tyrer, Penelope M. Webb, Nicolas Wentzensen, Emily White, Walter C. Willett, Alicja Wolk, Yin Ling Woo, Anna H. Wu, Yan Li, Drakoulis Yannoukakos, Georgia Chenevix‐Trench, Thomas A. Sellers, Paul D.P. Pharoah, Wei Zheng, Jirong Long

Bibliographic record

VenueCancer Research · 2018
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicEpigenetics and DNA Methylation
Canadian institutionsUniversité de MontréalCanadian Centre for Applied Research in Cancer ControlVancouver General HospitalAlberta Health ServicesVancouver Hospital and Health Sciences CentrePrincess Margaret Cancer CentreBC Cancer AgencyUniversity of British Columbia
FundersNational Cancer InstituteMedical Research CouncilCanadian Institutes of Health ResearchNational Institute for Health and Care ResearchJapan Society for the Promotion of ScienceNovo Nordisk FondenVanderbilt University Medical CenterCancer Research UKGovernment of CanadaWorld Health OrganizationEuropean CommissionNational Center for Advancing Translational SciencesWellcome TrustVanderbilt UniversityNational Center for Research ResourcesRoswell Park Cancer InstituteNational Institutes of HealthOvarian Cancer Research Fund
KeywordsDNA methylationCancerEpithelial ovarian cancerOvarian cancerMethylationOncologyMedicineBiologyInternal medicineDNAGynecologyGeneticsGeneGene expression

Abstract

fetched live from OpenAlex

Abstract DNA methylation is instrumental for gene regulation. Global changes in the epigenetic landscape have been recognized as a hallmark of cancer. However, the role of DNA methylation in epithelial ovarian cancer (EOC) remains unclear. In this study, high-density genetic and DNA methylation data in white blood cells from the Framingham Heart Study (N = 1,595) were used to build genetic models to predict DNA methylation levels. These prediction models were then applied to the summary statistics of a genome-wide association study (GWAS) of ovarian cancer including 22,406 EOC cases and 40,941 controls to investigate genetically predicted DNA methylation levels in association with EOC risk. Among 62,938 CpG sites investigated, genetically predicted methylation levels at 89 CpG were significantly associated with EOC risk at a Bonferroni-corrected threshold of P < 7.94 × 10−7. Of them, 87 were located at GWAS-identified EOC susceptibility regions and two resided in a genomic region not previously reported to be associated with EOC risk. Integrative analyses of genetic, methylation, and gene expression data identified consistent directions of associations across 12 CpG, five genes, and EOC risk, suggesting that methylation at these 12 CpG may influence EOC risk by regulating expression of these five genes, namely MAPT, HOXB3, ABHD8, ARHGAP27, and SKAP1. We identified novel DNA methylation markers associated with EOC risk and propose that methylation at multiple CpG may affect EOC risk via regulation of gene expression. Significance: Identification of novel DNA methylation markers associated with EOC risk suggests that methylation at multiple CpG may affect EOC risk through regulation of gene expression.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.363
Threshold uncertainty score0.618

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0010.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.001
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.065
GPT teacher head0.365
Teacher spread0.300 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations62
Published2018
Admission routes2
Has abstractyes

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