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Record W2920812796 · doi:10.1093/jcag/gwz006.085

A86 EXPLORING GENE-ENVIRONMENT INTERACTIONS IN CROHN’S DISEASE: THE EFFECT OF VITAMIN D DEFICIENCY ON INTESTINAL HOMEOSTASIS IN THE CONTEXT OF SUSCEPTIBILITY GENE NOD2L1007FS

2019· article· en· W2920812796 on OpenAlexaffabout
L. McGillis, Dana M. Bronte-Tinkew, Dana J. Philpott, Nicola L. Jones

Bibliographic record

VenueJournal of the Canadian Association of Gastroenterology · 2019
Typearticle
Languageen
FieldMedicine
TopicVitamin D Research Studies
Canadian institutionsHospital for Sick ChildrenUniversity of Toronto
Fundersnot available
KeywordsBiologyAutophagyContext (archaeology)Immune dysregulationVitamin A deficiencyImmunologyIntestinal epitheliumVitaminInflammatory bowel diseaseImmune systemEndocrinologyInternal medicineGeneticsMedicineDiseaseRetinolEpithelium

Abstract

fetched live from OpenAlex

Recent studies have implicated vitamin D deficiency in IBD susceptibility and outcomes. Although the mechanisms through which vitamin D affects IBD are not yet understood, emerging evidence from our lab and others demonstrate a role of vitamin D deficient signaling in dysregulating autophagy. In addition, vitamin D can modulate host immune responses and may play a role in regulating the stem cell niche. These effects of vitamin D have the potential to synergize with a major CD genetic risk factor, a frameshift mutation in the NOD2 gene (NOD2L1007fs or NOD2fs). NOD2fs results in a nonfunctional protein and causes dysregulation of inflammation and autophagy, pathways implicated in IBD pathogenesis. NOD2 mutations also effect the stem cell niche by causing dysregulation of Paneth cells, which represents an additional point of synergism for vitamin D deficiency and NOD2fs. We aim to determine if vitamin D deficiency in the context of the NOD2fs mutation results in alteration in intestinal homeostasis in vivo. We will also determine the effect of vitamin D on the intestinal epithelium using organoids generated from NOD2fs or wildtype mice. We hypothesize that vitamin D deficiency and the NOD2fs mutation will act synergistically to dysregulate autophagy and disrupt intestinal homeostasis, thereby promoting inflammation and driving susceptibility to CD. Mice homozygous for the NOD2fs CD susceptibility gene and wildtype littermates were fed either the vitamin D deficient (VDD) diet or matched control diet for 6 weeks. Ileum and colon sections were excised and lysates were used to assess the degree of autophagy between groups by western blotting for autophagy markers ATG16L1 and LC3II. Vitamin D ELISA of serum confirmed a significant reduction of vitamin D concentration in animals fed the VDD diet (p<0.0001). Unexpectedly, the NOD2fs mutation further reduced serum vitamin D concentration amongst animals on the control diet (p<0.0001) and animals on the VDD diet (p=0.051). The current results demonstrate reduction of ATG16L1 protein expression in ileal whole tissue lysates from animals with the NOD2fs mutation fed the VDD diet (p<0.05). This work will help to uncover gene-environment interactions involved in CD pathogenesis and will identify potential targetable pathways for development of novel therapies. The project has the potential for a translation through use of patient-derived organoids. CCCRestracomp (SickKids), Ontario Graduate Scholarship

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.999
Threshold uncertainty score0.010

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.001
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0030.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.020
GPT teacher head0.269
Teacher spread0.249 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2019
Admission routes2
Has abstractyes

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