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Record W2921993840 · doi:10.1093/jcag/gwz006.018

A19 ROLE OF LGR5 IN DCLK1 POSITIVE CELL-DERIVED COLITIS-ASSOCIATED COLON CANCER

2019· article· en· W2921993840 on OpenAlexaffabout
Alice E. Shin, Hayley Good, L Zhang, Elena N. Fazio, Philip M. Sherman, T C Wang, Samuel Asfaha

Bibliographic record

VenueJournal of the Canadian Association of Gastroenterology · 2019
Typearticle
Languageen
FieldMedicine
TopicHelicobacter pylori-related gastroenterology studies
Canadian institutionsHospital for Sick ChildrenUniversity of TorontoLawson Health Research InstituteSickKids FoundationWestern University
Fundersnot available
KeywordsLGR5Cancer researchBiologyInflammationCarcinogenesisColorectal cancerCancerMouse model of colorectal and intestinal cancerAdenomatous polyposis coliImmunology

Abstract

fetched live from OpenAlex

Colorectal cancer (CRC) is the second leading cause of cancer death in Canada, with the major risk factor being chronic inflammation. As such, patients with inflammatory bowel disease (IBD) are at an increased risk of CRC. Despite the clear association between inflammation and cancer, the mechanism by which colitis leads to CRC is still not well understood. Our recent work has focused on a colonic epithelial cell known as the tuft cell that uniquely expresses the protein doublecortin-like kinase 1 (Dclk1). Using Cre-dependent lineage tracing of Dclk1-expressing cells, we showed that Dclk1 labels long-lived quiescent cells in the colon that serve as a cellular origin of CRC upon colonic inflammation. In this study, we aim to explore the mechanism by which inflammation contributes to tuft cell cancer initiation. We hypothesized that colonic inflammatory insult leads to dedifferentiation of Dclk1+ tuft cells to a stem cell state susceptible to tumor initiation. To generate tamoxifen-inducible Cre transgenic mice that allow for Dclk1+ cell lineage tracing and cell-specific knock-out of the tumor suppressor adenomatous polyposis coli (APC), we first crossed our transgenic Dclk1-CreERT2 mice to both ROSA26-tdTomato and APCfl/fl mice (Dclk1/APCfl/fl). To examine the role of dedifferentiation in colonic tumor initiation, these mice were further crossed to Lgr5-DTR-eGFP mice (Lgr5DTR;Dclk1/APCfl/fl). These mice were given tamoxifen and DSS in order to induce tumorigenesis. Mice were administered diphtheria toxin (DT) for six weeks post DSS injury to ablate Lgr5-expressing cells and were compared to Lgr5DTR-negative control mice (Dclk1/APCfl/fl). Reverse transcription polymerase chain reaction (RT-PCR) analysis of mRNA levels revealed significantly reduced Lgr5, and increased RSPO1 and RSPO3 levels in colonic tissues of DSS-administered mice. Ablation of Lgr5+ cells post DSS-colitis significantly reduced the number of colonic tumors in our Dclk1/APCfl/fl mouse model, with no significant difference in tumor size. Lgr5-expressing cells were readily seen throughout colonic tumors arising from Dclk1+ cells. Interestingly, two weeks post DSS-induced colitis, rare Dclk1+ cells that co-expressed Lgr5 were also detected. Our data prove that upon DSS-induced colonic injury, Dclk1+ tuft cells express the stem cell marker Lgr5 prior to initiation of colonic tumorigenesis. These data suggest that dedifferentiation of Dclk1+ cells plays an important role in colitis-associated CRC and provide insight into the molecular mechanism by which Dclk1-derived colonic tumors arise. CAG, CIHRCRS

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.004

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.005
GPT teacher head0.221
Teacher spread0.216 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2019
Admission routes2
Has abstractyes

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