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Record W2943311090

The affect of androgens on fatty acid oxidation in prostate cancer cells

2008· article· en· W2943311090 on OpenAlexaff
Helen Lin, Pierre Laflamme, Inna Bryskin, Brian T. Wilson, Gurmit Singh, Jehonathan H. Pinthus

Bibliographic record

VenueCancer Research · 2008
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicCancer, Lipids, and Metabolism
Canadian institutionsMcMaster UniversityUniversity of Toronto
Fundersnot available
KeywordsBicalutamideAndrogenEndocrinologyProstate cancerInternal medicineAndrogen receptorChemistryCarnitineBeta oxidationFatty acidBiologyBiochemistryCancerMedicineHormone
DOInot available

Abstract

fetched live from OpenAlex

474 Epidemiological evidence suggests that dietary fat plays a role in the etiology of prostate cancer (PC). Oxidation of fatty acids (FA) results in the generation of reactive oxygen species (ROS), which have been postulated to play a key role in the initiation and progression of PC. The purpose of this study was to determine whether androgens, which stimulate the growth of PC, also play a role in fatty acid (FA) uptake and degradation and consequently increase mitochondrial ROS production. The model used compares the effect of a synthetic androgen (R1881) and an androgen receptor (AR) blocker (bicalutamide) on androgen responsive 22rv1 prostate cancer cells. Methods used were quantitative PCR, 14C-labeled long chain FA degradation assay and western blot analysis. We found that androgen supplementation increased the mRNA expression of the rate limiting enzyme in the fatty acid degradation Carnitine Palmitoyltransferase I (CPT1), in both liver and muscle isoforms and bicalutamide inhibited this phenomenon. Consequently, androgen supplementation increased the oxidation of long chain FA and this was reversed by bicalutamide. In addition, R1881 decreased the expression of Acetyl CoA Carboxylase (ACC) and its inactivated form phospho-ACC, an enzyme responsible for regulating the synthesis of the CPT1 inhibitor, malonyl-CoA. Subsequently we demonstrated that blockage of mitochondrial ROS generation by 2 different inhibitors- rotenone and thenoyltrifluoroacetone could eliminate the androgen induced generation of cellular ROS which is coupled to FA oxidation, bringing it to the same levels of PC cells that were deprived of androgens or treated with bicalutamide. Thus, we propose that the increase in ROS levels in PC cells is androgen regulated through the increased expression of CPT1 and decreased expression of ACC. Therefore, the FA that is taken up into PC cells is available for mitochondrial oxidation, a process that is also positively regulated by androgens, leading to the increased production of ROS that are associated with cancer cell proliferation and mutagenesis. In addition, the observed change in CPT1 and ACC expression may present a future target in reducing ROS levels for the purpose of PC prevention and therapy. Furthermore, dietary restrictions or anti-oxidants, also each have different inhibitory but complementary effects on the proposed mechanism.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.004
Threshold uncertainty score0.008

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.042
GPT teacher head0.364
Teacher spread0.322 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2008
Admission routes1
Has abstractyes

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