MétaCan
Menu
Back to cohort
Record W2998661164 · doi:10.1038/s41431-019-0563-6

TCTEX1D1 is a genetic modifier of disease progression in Duchenne muscular dystrophy

2020· article· en· W2998661164 on OpenAlexaff
Pietro Spitali, Irina Zaharieva, Stefan Böhringer, Monika Hiller, Amina Chaouch, Andreas Roos, C. Scotton, Mireille Claustres, Luca Bello, Craig M. McDonald, Eric P. Hoffman, Zaïda Koeks, H. Eka D. Suchiman, Sebahattin Çirak, Mariacristina Scoto, Mojgan Reza, Peter A.C. ’t Hoen, E. Niks, Sylvie Tuffery‐Giraud, Hanns Lochmüller, Alessandra Ferlini, Francesco Muntoni, Annemieke Aartsma‐Rus

Bibliographic record

VenueEuropean Journal of Human Genetics · 2020
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicMuscle Physiology and Disorders
Canadian institutionsOttawa HospitalUniversity of Ottawa
FundersFP7 HealthNational Institute on Disability and Rehabilitation ResearchNational Institute of Arthritis and Musculoskeletal and Skin DiseasesMedical Research CouncilU.S. Department of EducationGreat Ormond Street Hospital for ChildrenDeutsche ForschungsgemeinschaftU.S. Department of DefenseEuropean CommissionUniversity College LondonNational Institute for Health and Care ResearchU.S. Department of Health and Human ServicesNational Institutes of HealthNIHR Biomedical Research Centre, Royal Marsden NHS Foundation Trust/Institute of Cancer ResearchAssociation Française contre les Myopathies
KeywordsGeneticsDuchenne muscular dystrophyExome sequencingBiologyLocus (genetics)Candidate geneExomeDiseaseMuscular dystrophyAllelePhenotypeDystrophinSingle-nucleotide polymorphismBioinformaticsMedicineGeneGenotypeInternal medicine

Abstract

fetched live from OpenAlex

Duchenne muscular dystrophy (DMD) is caused by pathogenic variants in the DMD gene leading to the lack of dystrophin. Variability in the disease course suggests that other factors influence disease progression. With this study we aimed to identify genetic factors that may account for some of the variability in the clinical presentation. We compared whole-exome sequencing (WES) data in 27 DMD patients with extreme phenotypes to identify candidate variants that could affect disease progression. Validation of the candidate SNPs was performed in two independent cohorts including 301 (BIO-NMD cohort) and 109 (CINRG cohort of European ancestry) DMD patients, respectively. Variants in the Tctex1 domain containing 1 (TCTEX1D1) gene on chromosome 1 were associated with age of ambulation loss. The minor alleles of two independent variants, known to affect TCTEX1D1 coding sequence and induce skipping of its exon 4, were associated with earlier loss of ambulation. Our data show that disease progression of DMD is affected by a new locus on chromosome 1 and demonstrate the possibility to identify genetic modifiers in rare diseases by studying WES data in patients with extreme phenotypes followed by multiple layers of validation.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.804
Threshold uncertainty score0.564

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.016
GPT teacher head0.257
Teacher spread0.241 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations41
Published2020
Admission routes1
Has abstractyes

Explore more

Same venueEuropean Journal of Human GeneticsSame topicMuscle Physiology and DisordersFrench-language works237,207