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The myeloid lineage‐determining transcription factor PU.1 induces enhancer‐promoter looping that promotes IL‐1β enhancer and messenger RNA production in non‐myeloid melanoma cells

2019· article· en· W3015726723 on OpenAlexaffabout
Sung Ouk Kim, William Cho, Soon-Duck Ha

Bibliographic record

VenueThe FASEB Journal · 2019
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicRNA Interference and Gene Delivery
Canadian institutionsWestern University
Fundersnot available
KeywordsEnhancerTranscription factorChromatinPromoterMolecular biologyBiologyChromatin remodelingTranscription (linguistics)HistoneCell biologyChromatin immunoprecipitationEnhancer RNAsChemistryGene expressionGeneGenetics

Abstract

fetched live from OpenAlex

The DNA‐binding protein purine rich (PU.1) is a lineage determining transcription factor required for development of myeloid cells such as monocytes/macrophages. PU.1 is regarded as a pioneering transcription factor for macrophage differentiation due to its ability to bind “closed” genomic sites and initiate/maintain “open” chromatin state in macrophage‐restricted response genes. To date, however, precise mechanism of PU.1 in remodeling chromatin is yet to be elucidated. The non‐myeloid melanoma B16.BL6 cells response to the bacterial cell wall component lipopolysaccharide (LPS) and activate the transcription factor NF‐κB, but do not produce cytokines. Ectopic expression of PU.1 rendered these cells express the cytokine interleukin 1‐β (IL1b) in response to LPS. PU.1 induced chromatin remodeling that caused interaction between the IL1b promoter and enhancer (located ~10 kb upstream of the IL1b transcription start site) in non‐activated cells; upon activation, PU.1 recruited NF‐κB to these genomic regions. The N‐terminal domain (1–30) of PU.1 was important for both promoter‐enhancer looping and IL1b eRNA/mRNA production; whereas, the acidic and glutamine rich domains (33–100) were required for eRNA/mRNA production but dispensable for the DNA looping. eRNA production and histone acetylation were required for NF‐κB recruitment and IL1b mRNA expression, but not for the DNA looping. Altogether, this study indicates that PU.1 induces enhancer‐promoter looping that sequentially allows activation‐dependent histone acetylation, recruitment of NF‐κB and production of enhancer RNAs, all of which are required for an optimal IL1b production. Support or Funding Information The Natural Sciences and Engineering Research Council of Canada ‐ Discovery Grant (RGPIN‐2018‐05514) This abstract is from the Experimental Biology 2019 Meeting. There is no full text article associated with this abstract published in The FASEB Journal .

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.006

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.015
GPT teacher head0.238
Teacher spread0.223 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2019
Admission routes2
Has abstractyes

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