MétaCan
Menu
Back to cohort

Misoprostol attenuates hypoxia‐induced neonatal cardiomyocyte proliferation through Bnip3 and perinuclear calcium signaling

2020· article· en· W3017023395 on OpenAlexaffabout
Joseph W. Gordon, Matthew D. Martens, Jared T. Field, Donald Chapman, Chelsea Day, Richard Keijzer, Adrian R. West, Tammy L. Ivanco

Bibliographic record

VenueThe FASEB Journal · 2020
Typearticle
Languageen
FieldMedicine
TopicCardiac Ischemia and Reperfusion
Canadian institutionsUniversity of Manitoba
Fundersnot available
KeywordsHypoxia (environmental)Cell biologyBiologyHeart developmentMEF2CMisoprostolEndocrinologyInternal medicineMedicineChemistryGene expressionBiochemistryGeneEmbryonic stem cell

Abstract

fetched live from OpenAlex

Systemic hypoxia resulting from preterm birth, altered lung development, and cyanotic congenital heart disease is known to impede the regulatory and developmental pathways in the neonatal heart. Although the molecular mechanisms are still unknown, metabolic stressors resulting from neonatal hypoxia drive aberrant glycolysis and cardiomyocyte proliferation, which may be initially adaptive, but ultimately can program the heart for early life pathological remodeling. Recent evidence suggests the prostaglandin E1 analogue misoprostol is cytoprotective in the hypoxia‐exposed neonatal heart by promoting the alternative splicing of Bnip3, resulting in the generation of an isoform lacking the third exon (Bnip3ΔExon3) known as small Nip (sNip). Using a rodent model of neonatal hypoxia, in combination with rat primary ventricular neonatal cardiomyocytes and H9c2 cells, we sought to determine if misoprostol can prevent cardiomyocyte proliferation and what the key molecular mechanisms might be in this pathway. In cultured myocytes, molecular markers and fluorescent indicators of proliferation, such as cylcin‐D1 and PHH3, were significantly elevated with hypoxia exposure and were attenuated with misoprostol treatment (p<0.01). Moreover, hypoxia reduced mitochondrial respiration and increased cytosolic production of NADH, which were restored by misoprostol treatment. In addition, both sNip and misoprostol promoted cardiomyocyte maturation by increasing nuclear calcium accumulation, and drove the nuclear localization of NFATc3 (p<0.01). Gene expression studies identified that misoprostol and sNip reduced the expression of the proliferative MEF2C‐myocardin‐BMP10 pathway, while favoring expression of the cardiac maturation factors BMP2 and MEF2A (p<0.01). Furthermore, an siRNA targeting sNip restored hypoxia‐induced proliferation in misoprostol‐treated cells. Finally, we observed a significant increase in left ventricular nuclei number and PHH3 staining in hypoxia‐exposed rat pups, which were also attenuated with misoprostol treatment (p<0.05). These findings suggest a novel mechanism by which misoprostol promotes Bnip3 alternative splicing and myocyte maturation, thereby blocking hypoxia‐induced neonatal cardiomyocyte proliferation. Support or Funding Information This work is supported by the Natural Science and Engineering Research Council of Canada, the Heart and Stroke Foundation of Canada, and the Children’s Hospital Research Institute of Manitoba.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.003

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.033
GPT teacher head0.264
Teacher spread0.231 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2020
Admission routes2
Has abstractyes

Explore more

Same venueThe FASEB JournalSame topicCardiac Ischemia and ReperfusionFrench-language works237,207