MétaCan
Menu
← Back to cohort

A Mouse Model for Cerebro‐Costo‐Mandibular Syndrome (CCMS) with an Intronic Deletion in <i>SnrpB</i>

2020· article· en· W3017067098 on OpenAlexaffabout
Sabrina Alam, Jacek Majewski, Loydie Majewska

Bibliographic record

VenueThe FASEB Journal · 2020
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicRNA Research and Splicing
Canadian institutionsMcGill University
Fundersnot available
KeywordsExonBiologyMutantCraniofacialRNA splicingPenetranceGeneGeneticsRNAPhenotype

Abstract

fetched live from OpenAlex

SNRPB , an essential effector of the spliceosomal complex, is implicated in the splicing of mature mRNA and is mutated in a rare craniofacial disorder known as Cerebro‐costo‐mandibular syndrome (CCMS). Most CCMS patients carry point mutations in the highly conserved alternative exon 2 (AE2) of SNRPB , leading to increased inclusion of a pre‐termination codon (PTC) and reduced levels of the protein‐coding transcript. Given that SNRPB is implicated in all steps of splicing, it was thought that its requirement would be equivalent in all cells and tissues. However, CCMS patients predominantly present with micrognathia and rib defects with variable penetrance. We hypothesize that a subset of transcripts is highly sensitive to the levels of SNRPB during development and is tissue specific. Here, we are generating a mouse model for CCMS to uncover those transcripts that play an essential role in craniofacial development. Using the CRISPR/Cas9 gene‐editing system, we developed a mutant mouse line with a 61 base‐pair (bp) intronic deletion upstream of the AE2. Interestingly, a subset of the heterozygous and homozygous embryos and pups for this deletion show morphological abnormalities in the face, head, ribs, and limbs. Moreover, a significant percentage of the mutants with the 61‐bp deletion die in their first year of life. A subgroup of the heterozygous and homozygous mutant embryos show increased inclusion of the PTC containing AE2 and reduced SNRPB protein levels. We presume that this predominant inclusion of the AE2 induces non‐sense mediated decay resulting in decreased levels of SNRPB. As our data suggest that the 61 bp intronic sequence plays a critical role in regulating SNRPB levels in mice, we are currently investigating if similar intronic sequences in intron 2 of human SNRPB are involved in the preferential splicing of AE2. In addition, we generated a SnrpB conditional knockout mouse model, which will be used in parallel with the deletion model to investigate the cell‐type‐specific roles of SNRPB during craniofacial development. Our novel model for CCMS will further our understanding of the role of SNRPB in craniofacial development. Support or Funding Information Canadian Institutes of Health Research (CIHR)

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.007
Threshold uncertainty score0.022

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.000
Meta-epidemiology (narrow)0.0010.001
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0020.001
Science and technology studies0.0010.001
Scholarly communication0.0010.001
Open science0.0010.001
Research integrity0.0020.003
Insufficient payload (model declined to judge)0.0070.002

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.019
GPT teacher head0.254
Teacher spread0.235 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2020
Admission routes2
Has abstractyes

Explore more

Same venueThe FASEB Journal→Same topicRNA Research and Splicing→French-language works237,207→