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Record W3035507687 · doi:10.2337/db20-1805-p

1805-P: Dietary Nitrate Prevents High-Fat Diet-Induced Glucose Intolerance and Liver-Specific Reactive Oxygen Species Emission

2020· article· en· W3035507687 on OpenAlexaboutno aff
Geneviève J. DesOrmeaux, Heather L. Petrick, Henver S. Brunetta, Graham P. Holloway

Bibliographic record

VenueDiabetes · 2020
Typearticle
Languageen
FieldMedicine
TopicDiet and metabolism studies
Canadian institutionsnot available
Fundersnot available
KeywordsInternal medicineEndocrinologyRespirationReactive oxygen speciesLipid oxidationInsulin resistanceChemistryLipid metabolismCellular respirationMitochondrial ROSMitochondrionBiologyBiochemistryInsulinAntioxidantMedicine

Abstract

fetched live from OpenAlex

Introduction: The liver is particularly susceptible to the detrimental effects of a high fat diet (HFD), and rapidly develops insulin resistance. While the underlying mechanisms associated with hepatic insulin resistance are not fully understood, mitochondria play a crucial role in regulating lipid metabolism and redox balance. Recently, dietary nitrate was shown to attenuate HFD-induced glucose intolerance, however the mechanism(s) remains unknown. Therefore, in the present study we examined the possibility that dietary nitrate increased mitochondrial lipid-supported respiration and/or attenuated reactive oxygen species (ROS) emission. Methods: Male C57Bl/6J mice (n = 22) were randomly assigned to consume a control diet (10% fat), HFD (60% fat), or HFD with 4mM sodium nitrate in drinking water for 8 weeks. Whole body glucose tolerance and energy expenditure (indirect calorimetry) were determined, and mitochondrial respiration and reactive oxygen species (ROS) emission were assessed in liver samples. Results: Compared to control animals, the consumption of HFD increased whole body fat oxidation and body mass, and induced glucose intolerance. The consumption of dietary nitrate did not affect HFD-mediated changes in energy expenditure, rates of fat oxidation or body mass, but nevertheless attenuated the induction of glucose intolerance. Within the liver, HFD-feeding in the presence or absence of dietary nitrate, did not alter pyruvate or lipid (palmitoyl-CoA) supported respiration, or the sensitivity to malonyl-CoA-mediated inhibition of lipid-supported respiration. In contrast, while HFD increased the endogenous ROS production, nitrate prevented this response. Conclusion: These data demonstrate that nitrate supplementation attenuated the HFD-mediated induction of whole-body glucose intolerance in association with a reduction in ROS emission within the liver. Future research needs to determine causality between these observations. Disclosure G. DesOrmeaux: None. H.L. Petrick: None. H. Brunetta: None. G. Holloway: None. Funding Natural Sciences and Engineering Research Council of Canada (400362)

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.005
Threshold uncertainty score0.016

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0000.000
Science and technology studies0.0000.001
Scholarly communication0.0000.000
Open science0.0010.000
Research integrity0.0010.002
Insufficient payload (model declined to judge)0.0050.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.034
GPT teacher head0.245
Teacher spread0.211 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2020
Admission routes1
Has abstractyes

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