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Record W3108057420 · doi:10.1093/clinchem/hvaa243

Commentary on A Rare Cause of Virilization, Short Stature, and Hypertension

2020· letter· en· W3108057420 on OpenAlexaff
Fady Hannah‐Shmouni, Andrew Don-Wauchope

Bibliographic record

VenueClinical Chemistry · 2020
Typeletter
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicSexual Differentiation and Disorders
Canadian institutionsMcMaster University
Fundersnot available
KeywordsVirilizationShort statureMedicinePediatricsEndocrinologyInternal medicineHormoneAndrogen

Abstract

fetched live from OpenAlex

The patient described presented with acute illness as a result of poor compliance with medication either due to poor understanding of her condition by her or the people caring for her and/or due to the inability to acquire her medications. The consequences are probably not yet fully realized. Her mineralocorticoid hypertension is severe and the risk for complications is high. The clinical and laboratory features are fairly classic for 11β-hydroxylase deficiency (11-OHD), manifesting with virilization, short stature, and severe hypertension. She presented with a hypertensive hypokalemic disorder following interruption of steroids, due to apparent mineralocorticoid excess from 11-deoxycorticosterone, and possibly other steroid precursors, providing an important diagnostic clue to 11-OHD (1). 11-OHD is autosomal recessive and caused by pathogenic variants in CYP11B1 (8q24.3). This gene consists of 9 exons that are approximately 40-kb from the highly homologous CYP11B2 (2). Unlike nonclassic Congenital Adrenal Hyperplasia (CAH) due to CYP21A1 (prevalence of 1 in 200 regardless of ethnicity) (2), 11-OHD is rare, with highest prevalence among Moroccan Jews due to a founder effect. The patient and her sibling were compound heterozygotes carrying 2 different disease-causing variants, as seen in most cases of CAH, with the phenotype defined by the variant retaining the most enzyme activity. Since both parents are presumed to be carriers, the genetic risk in each offspring is 25% for CAH, and 50% for carrier. The goals of therapy for 11-OHD should be achieved with regular clinical assessments. Comorbidities to address includes degree of virilization, growth velocity, control of hypertension and metabolic syndrome, fertility, bone health, adrenal insufficiency/sick day teaching and genetic counseling (1). Metabolic control with glucocorticoid replacement in the form of hydrocortisone is preferred for reduction of excess androgens. Hypertension is best managed with a mineralocorticoid receptor antagonist (2). During acute illness, supraphysiologic glucocorticoid therapy with or without mineralocorticoid replacement for electrolyte imbalance is warranted. All authors confirmed they have contributed to the intellectual content of this paper and have met the following 4 requirements: (a) significant contributions to the conception and design, acquisition of data, or analysis and interpretation of data; (b) drafting or revising the article for intellectual content; (c) final approval of the published article; and (d) agreement to be accountable for all aspects of the article thus ensuring that questions related to the accuracy or integrity of any part of the article are appropriately investigated and resolved. No authors declared any potential conflicts of interest.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.002
metaresearch head score (Gemma)0.021
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Case report · Consensus signal: none
GenreCandidate signal: Commentary · Consensus signal: Commentary
Teacher disagreement score0.047
Threshold uncertainty score0.023

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0020.021
Meta-epidemiology (narrow)0.0010.001
Meta-epidemiology (broad)0.0020.001
Bibliometrics0.0010.001
Science and technology studies0.0030.002
Scholarly communication0.0020.002
Open science0.0020.001
Research integrity0.0470.027
Insufficient payload (model declined to judge)0.0050.004

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.050
GPT teacher head0.325
Teacher spread0.275 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designCase report
Domainnot available
GenreCommentary

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2020
Admission routes1
Has abstractno

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