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Record W3111632648 · doi:10.1093/neuonc/noaa215.280

EPCO-01. LUNG ADENOCARCINOMA BRAIN METASTASIS PREDICTION, PREVENTION, AND NON-INVASIVE DIAGNOSIS USING METHYLATION SIGNATURES WITHIN TISSUE AND CIRCULATING TUMOUR DNA

2020· article· en· W3111632648 on OpenAlexaff
Jeffrey Zuccato, Yasin Mamatjan, Vikas Patil, Farshad Nassiri, Mathew Voisin, Kenneth Aldape, Gelareh Zadeh

Bibliographic record

VenueNeuro-Oncology · 2020
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicRNA modifications and cancer
Canadian institutionsPrincess Margaret Cancer CentreUniversity Health NetworkUniversity of Toronto
Fundersnot available
KeywordsDNA methylationMethylationAdenocarcinomaBrain metastasisLung cancerOncologyMetastasisDifferentially methylated regionsCancer researchBiologyStage (stratigraphy)CohortCancerPathologyInternal medicineMedicineDNAGeneGeneticsGene expression

Abstract

fetched live from OpenAlex

Abstract BACKGROUND One quarter of lung adenocarcinoma (LUAD) patients develop brain metastases (BM) and experience a poorer median survival of 12 months despite treatment. Clinical variables do not robustly predict who will develop BM and targeted preventative treatments are limited. Tumour DNA methylation signatures predict outcomes in other cancers and can be detected in circulating tumour DNA (ctDNA). This work predicts BM development from LUAD using methylation data, identifies novel potential treatment targets to prevent metastases, and detects LUAD-BM ctDNA non-invasively. METHODS DNA methylation profiling was undertaken on N=124 LUAD tumours. A gradient boosted regression model built on differentially methylated CpGs (DMCs) between tumours with and without BM in 70% of samples was validated in an independent 30% testing cohort. Nine paired BM samples were profiled and DMCs between their corresponding LUAD tissue were identified along with copy number (CN) alterations. A total of 47 LUAD-BM plasma samples underwent sequencing of immunoprecipitated methylated ctDNA and differentially methylated regions (DMRs) between LUAD-BM and intrinsic brain lesions were identified. RESULTS The methylation-based model significantly predicted time to brain metastasis development within the testing cohort independently from cancer stage in a multivariate analysis (HR=4.3, 95%CI 1.1–17, p=0.038). Genes/pathways involved in the process of brain metastasis were identified through assessment of 83K DMCs (FDR< 0.2, mean difference >|0.1|) between paired samples as well as the CN losses found in chromosome 12q/19 of BM samples. A total of 5.5K DMRs were identified that distinguish BM samples from gliomas or primary CNS lymphomas (FDR< 0.05, logFC >1). CONCLUSIONS DNA methylation signatures in lung adenocarcinomas predict brain metastasis development independently from prognostic clinical factors. Genes and pathways involved in metastasis were identified as novel potential therapeutic targets. Methylated circulating tumor DNA signatures differentiate lung brain metastases from other ring-enhancing brain lesions and may have potential for non-invasive diagnosis.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.002
metaresearch head score (Gemma)0.004
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.007
Threshold uncertainty score0.018

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0020.004
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0010.001
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0050.002

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.025
GPT teacher head0.298
Teacher spread0.272 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2020
Admission routes1
Has abstractyes

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