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Record W3111905501 · doi:10.1002/alz.040613

TGF344‐AD rats exhibit cognitive reserve despite continual Aβ and tau accumulation

2020· article· en· W3111905501 on OpenAlexaff
Christopher D. Morrone, Aaron Y. Lai, Mary Hill, JoAnne McLaurin

Bibliographic record

VenueAlzheimer s & Dementia · 2020
Typearticle
Languageen
FieldMedicine
TopicAlzheimer's disease research and treatments
Canadian institutionsUniversity of TorontoSunnybrook Hospital
Fundersnot available
KeywordsNeurodegenerationCognitive reserveCognitive declineNeuroscienceEntorhinal cortexHippocampusCognitionDiseasePsychologyDementiaAlzheimer's diseaseMedicinePathologyCognitive impairment

Abstract

fetched live from OpenAlex

Abstract Background Patient to patient variability in Alzheimer’s disease progression complicates treatment efficacy. Furthermore, the presence of Aβ pathology does not always indicate cognitive impairments. Epidemiological evidence of cognitive reserve suggests some individuals exhibit enhanced neuronal and behavioural resilience, protecting against neurodegeneration and cognitive decline. However, Alzheimer’s disease models typically exhibit a correlation between accumulation of pathology and behavioural impairments, making cognitive reserve difficult to investigate preclinically. Method TgF344‐AD rats recapitulate the amyloid cascade with a long‐term accumulation of Aβ and tau pathology leading to significant neurodegeneration and behavioural impairments. TgF344‐AD rats were assessed for cognitive and neuropathological alterations compared to age‐matched non‐transgenic littermates, across disease progression at 4, 6, 9, 12 and 15 months of age. Latency to escape and complexity of search strategies utilized in the Barnes maze spatial and reversal phases were assessed to determine learning, long‐term memory, and executive function. Neuropathological assessment included GABAergic and total neuronal counts, and quantification of Aβ and tau pathology. Result TgF344‐AD rats demonstrate deficits in spatial memory and executive function beginning at 6 months which are not exacerbated at 9 or 12 months, but are at 15 months of age. GABAergic and total neuronal counts in the hippocampus and entorhinal cortex of TgF344‐AD rats exhibit a similar trend, with deficits not progressing between 9 and 12 months of age. Conversely, Aβ and tau pathology accumulate continually across disease progression. Non‐transgenic rats exhibit slight, yet consistent age‐related decline in cognitive and neuronal measures. Conclusion Disease‐bearing TgF344‐AD rats exhibit cognitive reserve before robust cognitive decline at 15 months, despite consistent increases in Aβ and tau deposition. Maintenance, and in certain hippocampal subregions a recovery, of neuronal loss at 12 months support this observation, and suggest the involvement of compensatory mechanisms such as hippocampal neurogenesis. Understanding the mechanisms underlying cognitive reserve in Alzheimer’s disease will help explain patient variability in disease progression and elucidate novel regenerative therapeutic targets.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.006

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.088
GPT teacher head0.355
Teacher spread0.268 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2020
Admission routes1
Has abstractyes

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