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Record W3112375597 · doi:10.1038/s41598-020-78471-3

Cellular correlates of cortical thinning throughout the lifespan

2020· article· en· W3112375597 on OpenAlexafffund
Didac Vidal‐Piñeiro, Nadine Parker, Jean Shin, Leon French, Håkon Grydeland, Andrea Parolin Jackowski, Athanasia M. Mowinckel, Yash Patel, Zdenka Pausová, Giovanni Abrahão Salum, Øystein Sørensen, Kristine B. Walhovd, Tomáš Paus, Anders M. Fjell

Bibliographic record

VenueScientific Reports · 2020
Typearticle
Languageen
FieldMedicine
TopicDiet and metabolism studies
Canadian institutionsHospital for Sick ChildrenCentre for Addiction and Mental HealthSickKids FoundationHolland Bloorview Kids Rehabilitation HospitalUniversity of Toronto
FundersNational Institute of Biomedical Imaging and BioengineeringCanadian Institutes of Health ResearchNational Institutes of HealthNorges ForskningsrådGenentechIXICOH. Lundbeck A/SServierEisaiCoordenação de Aperfeiçoamento de Pessoal de Nível SuperiorEuropean Research CouncilNorthern California Institute for Research and EducationEli Lilly and CompanyU.S. Department of DefenseMeso Scale DiagnosticsUniversitetet i OsloF. Hoffmann-La RocheUniversity of Southern CaliforniaPfizerBiogenBioClinicaNovartis Pharmaceuticals CorporationHeart and Stroke Foundation of CanadaBristol-Myers SquibbNational Institute on AgingCommonwealth Scientific and Industrial Research OrganisationFoundation for the National Institutes of Health
KeywordsThinningNeuroscienceBiologyEcology

Abstract

fetched live from OpenAlex

Cortical thinning occurs throughout the entire life and extends to late-life neurodegeneration, yet the neurobiological substrates are poorly understood. Here, we used a virtual-histology technique and gene expression data from the Allen Human Brain Atlas to compare the regional profiles of longitudinal cortical thinning through life (4004 magnetic resonance images [MRIs]) with those of gene expression for several neuronal and non-neuronal cell types. The results were replicated in three independent datasets. We found that inter-regional profiles of cortical thinning related to expression profiles for marker genes of CA1 pyramidal cells, astrocytes and, microglia during development and in aging. During the two stages of life, the relationships went in opposite directions: greater gene expression related to less thinning in development and vice versa in aging. The association between cortical thinning and cell-specific gene expression was also present in mild cognitive impairment and Alzheimer's Disease. These findings suggest a role of astrocytes and microglia in promoting and supporting neuronal growth and dendritic structures through life that affects cortical thickness during development, aging, and neurodegeneration. Overall, the findings contribute to our understanding of the neurobiology underlying variations in MRI-derived estimates of cortical thinning through life and late-life disease.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.001
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Not applicable · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.466
Threshold uncertainty score0.236

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0010.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.001
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.031
GPT teacher head0.291
Teacher spread0.260 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designNot applicable
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations160
Published2020
Admission routes2
Has abstractyes

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