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A Novel Mitochondrial Fission Inhibitor Ameliorates DSS and DNBS Induced Murine Colitis

2018· article· en· W3173475413 on OpenAlexaffabout
Luke Goudie, Nicole L. Mancini, Karen Bloté, Arthur Wang, Derek M. McKay, Jane Shearer

Bibliographic record

VenueThe FASEB Journal · 2018
Typearticle
Languageen
FieldMedicine
TopicLiver Disease Diagnosis and Treatment
Canadian institutionsUniversity of Calgary
Fundersnot available
KeywordsColitisFIS1Inflammatory bowel diseaseMitochondrial fissionChemistryMitochondrionPharmacologyImmunologyBiologyInternal medicineBiochemistrymitochondrial fusionMedicineDiseaseMitochondrial DNAGene

Abstract

fetched live from OpenAlex

Significance Inflammatory bowel disease (IBD) encompasses a group of disorders that involve an exaggerated immune response to intestinal microbes. Maintenance of intestinal epithelial barrier function is highly dependent on normal mitochondrial networks. When the barrier is compromised, excessive pathological fission ensues via the enzyme dynamin related protein‐1 (DRP1) and its interaction with the receptor fission protein‐1 (Fis1), creating fragmented networks of mitochondria. Fragmented mitochondrial networks have impaired respiratory capacity, generate elevated reactive oxygen species and exacerbate inflammation Objective To determine whether a novel peptide, P110, can mitigate dextran sodium sulfate (DSS) and dinitrobenzene sulfonic acid (DNBS) induced colitis by inhibiting DRP1 and Fis1 mediated fission in murine models. Design Hypothesising that elevated mitochondrial fission occurs in DSS and DNBS colitis, male Balb/c mice were split into control (n=15), P110 (3 mg/kg/d, intraperitoneally)(n=8), DSS (5% (w/v), 5 days + 3 days water)(n=16) and DSS+P110 (n=16). For the DNBS protocol, male Balb/c mice were divided into control (n=8), DNBS (3 mg/kg/d, intraperitoneally)(n=11) and DNBS+P110 (3 mg/kg/d, intraperitoneally)(n=8). Experimental measures included: body mass, colon length, macroscopic disease score, cross‐sectional histopathology and colonic motility. Results Upon necropsy DSS and DNBS treated mice displayed the characteristic signs of colitis associated with these models. Disease was substantially less in both DSS+P110 and DNBS+P110 treated mice as gauged by: (i) percent weight loss (ii) macroscopic disease score and (iii) colon shortening (p<0.05). Functional improvements in colonic motility were seen in P110 treated mice when compared to DSS alone (p<0.05). Analysis of histopathology on H&E stained sections of mid‐colon revealed significant improvement in DNBS mice with P110 (p<0.05). P110 was also found to improve survival rate of mice, compared to DNBS alone. Conclusions Systemic administration of a selective inhibitor of mitochondrial fission (P110) reduced the severity of disease in two different, commonly used murine models of colitis. Future studies are required to define the mechanism of P110's action in terms of the target cell in IBD (e.g. epithelium, nerve, macrophage). We conclude that inhibition of DRP1 and Fis1 interaction provides a novel approach to mitigating gut barrier deterioration in IBD Support or Funding Information Crohn's and Colitis Canada. The authors wish to thank Daria Mochly‐Rosen for donating P110. This abstract is from the Experimental Biology 2018 Meeting. There is no full text article associated with this abstract published in The FASEB Journal .

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.005

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.025
GPT teacher head0.277
Teacher spread0.251 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2018
Admission routes2
Has abstractyes

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