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Mechanism of obesity suppression by adipose tissue creatine energetics

2018· article· en· W3175620243 on OpenAlexafffundabout
Lawrence Kazak, Gina Z. Lu, Bruce M. Spiegelman

Bibliographic record

VenueThe FASEB Journal · 2018
Typearticle
Languageen
FieldMedicine
TopicAdipose Tissue and Metabolism
Canadian institutionsMcGill University
FundersNational Institute of Diabetes and Digestive and Kidney DiseasesCanadian Institutes of Health Research
KeywordsThermogenesisInternal medicineEndocrinologyCreatineBrown adipose tissueThermogeninAdipose tissueMitochondrionCreatine kinaseAdipocyteBiologyChemistryMedicineBiochemistry

Abstract

fetched live from OpenAlex

The increase in obesity worldwide has led to a surge in the prevalence of type 2 diabetes, cardiovascular disease and many cancers. The capacity of thermogenic (brown and beige) fat to dissipate chemical energy as heat offers tremendous potential to combat obesity. Exploiting thermogenesis to regulate body weight requires a precise molecular understanding of its control at the level of mitochondria. We have previously demonstrated that creatine drives energy expenditure in adipose tissue by stimulating a futile cycle of mitochondrial ATP turnover. Our recent findings show that genetic deletion of the rate‐limiting enzyme of creatine synthesis (Gatm) results in rapid‐onset obesity due to the suppression of elevated energy expenditure that occurs in response to high calorie feeding. This is paralleled by a blunted capacity for β3‐adrenergic activation of metabolic rate. Moreover, abolishing adipocyte creatine uptake, by deleting the creatine transporter (CrT), similarly impairs adrenergic thermogenesis and causes obesity. These results provide strong in vivo genetic support for a role of adipose tissue creatine metabolism in the activation of energy expenditure and the defense against diet‐induced obesity. However, the mechanism underlying creatine‐dependent thermogenesis is not fully understood. We have now identified the primary Creatine Kinase (CK) isoform that controls thermogenic respiration in brown fat. Using quantitative proteomics from purified brown adipocytes and brown adipocyte mitochondria, we find brain‐type CK (B‐CK) to be the most abundant CK in murine brown fat. Surprisingly a subset of B‐CK is targeted to mitochondria. Mitochondria from primary brown adipocytes have robust capacity for creatine‐dependent thermogenic respiration, and knockdown of B‐CK impairs this process. Notably, B‐CK is the most abundance CK isoform in purified human subcutaneous adipocytes and its expression is significantly lower in insulin‐resistant compared to insulin‐sensitive patients. Therefore, B‐CK is the first molecular handle that should result in the identification of additional components regulating creatine‐dependent thermogenesis. In addition, adipose tissue creatine energetics is correlated with healthy glucose homeostasis in humans. Support or Funding Information Canadian Institutes of Health Research postdoctoral fellowship and NIDDK, NIH Pathway to Independence (K99/R00), K99DK114528 (to L.K.) NIH grant (DK31405) and the JPB Foundation (to B.M.S.). This abstract is from the Experimental Biology 2018 Meeting. There is no full text article associated with this abstract published in The FASEB Journal .

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.004

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.001
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.014
GPT teacher head0.274
Teacher spread0.259 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2018
Admission routes3
Has abstractyes

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