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Induction of asthma causes sensitization of the carotid bodies to lysophosphatidic acid

2019· article· en· W3175991509 on OpenAlexaffabout
Nicholas Jendzjowsky, Arijit Roy, Richard J. A. Wilson

Bibliographic record

VenueThe FASEB Journal · 2019
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicSphingolipid Metabolism and Signaling
Canadian institutionsUniversity of Calgary
Fundersnot available
KeywordsLysophosphatidic acidOvalbuminBronchoconstrictionAsthmaInternal medicineSensitizationEndocrinologyMedicineMethacholineTRPV1ReflexChemistryReceptorImmunologyLungTransient receptor potential channelImmune systemRespiratory disease

Abstract

fetched live from OpenAlex

Previously, we demonstrated an important role for the carotid bodies in asthmatic bronchoconstriction. Specifically, we showed that an allergen‐induced increase in circulating lysophosphatidic acid (LPA) in asthmatic rats activates LPA receptors (LPAr) in the carotid bodies and post‐ganglionic TRPV1 channels, provoking vagal activity and thereby triggering acute bronchoconstriction (Jendzjowsky et al. 2018 Nat Comm. 9, 4030). Here we report that induction of asthma causes molecular and functional changes to the carotid bodies that sensitize them to LPA. Experiments were carried out using the standard ovalbumin‐sensitized brown Norway rat model of asthma. Carotid bodies and petrosal ganglia were extracted 3 hours after the third ovalbumin challenge in asthmatic (OVA) or third saline challenge in naïve (N) rats. Using qPCR (Δctt OVA vs N), LPAr expression was increased in the carotid body (LPAr1: 556x; LPAr2: 20x; LPAr3: 2x; LPAr4: 150x; LPAr5 only detected in OVA: LPAr6: 175x; TRPV1 not detected, as demonstrated previously Roy et al. 2012 J Appl Physiol 112:212–224) whereas LPAr 1–6 and TRPV1 were similarly expressed in petrosal ganglia of OVA and N rats. Using the perfused ex vivo carotid body preparation, baseline normalized carotid sinus nerve responses to increasing doses of LPA activity, were significantly augmented in OVA compared to N rats (2.5uM‐ OVA: 1.12 ± 0.01 N: 1.05 ± 0.02, p=0.162; 5uM: OVA: 1.23 ± 0.03 N:1.10 ± 0.02, p=0.009; 10uM: OVA: 1.40 ± 0.07 N: 1.17 ± 0.03, p<0.001). In contrast, carotid body activity in response to hypoxia was not different between OVA (2.54 ± 0.47) and N (1.95 ± 0.10) rats (p=0.243). Our data suggest that asthmatic carotid bodies have enhanced responsiveness to LPA, independent of hypoxic sensitivity. These changes likely contribute to the acute allergen‐induced bronchoconstriction which is the hallmark of asthma that appears to be mediated, in part, by a novel carotid body‐vagal‐lung neuronal pathway. Targeting carotid body LPA signalling may reduce asthmatic bronchoconstriction and provide new pharmaceutical targets for inflammatory lung disease. Support or Funding Information This project was funded by The Lung Association and Canadian Institute for Health Research. This abstract is from the Experimental Biology 2019 Meeting. There is no full text article associated with this abstract published in The FASEB Journal .

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.006

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.001
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.008
GPT teacher head0.223
Teacher spread0.214 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2019
Admission routes2
Has abstractyes

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