MétaCan
Menu
← Back to cohort

Mouse models of human craniofacials spliceosomopathies: Are they neurocristopathies?

2019· article· en· W3177237293 on OpenAlexaffabout
Loydie A. Jerome‐Majewska, Marie‐Claude Beauchamp, Anissa Djedid, Kévin Daupin, Kayla Clokie, Shruti Kumar, Sabrina Alam, Jacek Majewski

Bibliographic record

VenueThe FASEB Journal · 2019
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicRNA Research and Splicing
Canadian institutionsMcGill UniversityMcGill Genome CentreMcGill University Health Centre
Fundersnot available
KeywordsBiologyIntronGeneticsRNA splicingExonMolecular biologyCell biologyGeneRNA

Abstract

fetched live from OpenAlex

Splicing, the removal of introns from pre‐messenger RNA is an essential step for expression of most genes in multicellular organisms and for expanding the number of proteins coded by genomes. Recently, exome sequencing revealed that mutations in splicing factors and small nuclear ribonucleoprotein particles (snRNPs) which form core components of the spliceosome, are responsible for craniofacial malformations. Our group used in situ hybridization to examine expression of three core components of the spliceosome: Eftud2 – mutated in patients with mandibulofacial dysostosis, Guion‐Almeida type (MFDGA); SnrpB – responsible for Cerebrocostomandibular Syndrome (CMS); and Sf3b4 which is responsible for Nager syndrome. Herein, we report expression of Eftud2, Sf3b4 and SnrpB during craniofacial development. In addition, we used CRISPR/Cas9 to generate mice with deletion ( Eftud2 del) and conditional mutation of exon 2 of Eftud2 (Eftud2 flox) . At embryonic days (E) 7.5 and 8.5, Eftud2 was highly expressed in ectodermal and mesodermal components of the future craniofacial region, by E9.5 expression was also found in the body wall and developing heart. Eftud2 del heterozygous mice were viable and fertile, though these embryos showed reduced levels of Eftud2 mRNA and protein. In contrast, Eftud2 del homozygous mutant embryos arrest at E3.5 and failed to grow and hatch ex vivo . To examine the requirement for Eftud2 in neural crest cells, the Wnt1‐Cre2 transgenic line was used to delete exon 2 of Eftud2 specifically in that lineage. Eftud2 flox homozygous mutant embryos carrying the Wnt1‐Cre2 transgene displayed hypoplasia of the midbrain and pharyngeal arches starting at E9.5. By E11.5, most embryos also had an open neural tube and all embryos showed exencephaly at E14.5. Cartilage preparations revealed an absence of cartilage in the head, reduction/or absence of Meckel's cartilage, and abnormal inner ear development. Since deletion of exon 2 is predicted to generate a truncated protein with partial function, our data suggest that normal levels of Eftud2 is crucial in neural crest cells for normal craniofacial development. Future studies are focused on elucidating the molecular and transcriptional basis of MFDM using this mouse model. Support or Funding Information Canadian Institute of Health Research (MOP#142452) This abstract is from the Experimental Biology 2019 Meeting. There is no full text article associated with this abstract published in The FASEB Journal .

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.006
Threshold uncertainty score0.021

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.000
Meta-epidemiology (narrow)0.0010.001
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0020.001
Science and technology studies0.0010.001
Scholarly communication0.0010.001
Open science0.0010.001
Research integrity0.0020.002
Insufficient payload (model declined to judge)0.0060.002

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.020
GPT teacher head0.268
Teacher spread0.248 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2019
Admission routes2
Has abstractyes

Explore more

Same venueThe FASEB Journal→Same topicRNA Research and Splicing→French-language works237,207→