MétaCan
Menu
Back to cohort

Nicotinic acetylcholine receptor silent agonists modulate inflammation

2019· article· en· W3177272872 on OpenAlexafffundabout
Alain R. Simard, Katrin Richter, Danika Roy, Eduardo Soto Espinosa, Patrick Roy, Jean‐Rémi Godin, Veronika Grau, Marta Quadri, Nicole A. Horenstein, Roger L. Papke

Bibliographic record

VenueThe FASEB Journal · 2019
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicNicotinic Acetylcholine Receptors Study
Canadian institutionsNOSM UniversityLaurentian UniversityUniversité de Moncton
FundersNational Institutes of HealthFondation de la recherche en santé du Nouveau-Brunswick
KeywordsMethyllycaconitineCytokineMecamylamineNicotinic agonistAgonistChemistryPharmacologyMonocyteReceptorInflammationNicotinic acetylcholine receptorImmunologyBiologyBiochemistry

Abstract

fetched live from OpenAlex

Nicotinic acetylcholine receptors (nAChRs) have been increasingly studied for their immune regulatory properties. nAChRs are best known to function as ligand‐gated ion channels in neurons, however, recent evidence suggests that nicotine modulates inflammation by desensitizing non‐neuronal nAChRs, rather than by inducing channel opening. The objective of this study was to determine whether nAChR‐dependent anti‐inflammatory properties are due to channel desensitization. To this end, we made use of various nAChR ligands, including: 1) nicotine, a nAChR non‐selective agonist that also desensitizes the receptors; 2) AR‐R17779, an α7 nAChR subtype‐selective agonist; 3) α‐bungarotoxin, an α7 and α9 nAChR‐selective competitive antagonist, 4) mecamylamine, a non‐selective and non‐competitive antagonist; 5) N,N‐diethyl‐N’‐phenylpiperazine (DEPP)‐based silent agonists that selectively induce the desensitized state of α7 nAChR while producing little or no channel opening. We first evaluated the effects of these various nAChR ligands on the modulation of mouse and human monocyte/macrophage numbers, phenotype and cytokine production. Monocyte phenotype, cellular viability, proliferation and cytokine production were assessed by flow cytometry. In bone marrow‐derived monocyte/macrophage (BMDM) cell cultures, nicotine, mecamylamine and silent agonists reduced pro‐inflammatory monocyte numbers by affecting their viability and proliferation. The silent agonists also significantly reduced both LPS‐ and ATP‐induced cytokine production by mouse BMDMs. In human peripheral blood mononuclear cells (PBMCs), the silent agonists tested had modest effects on monocyte numbers and phenotype. However, LPS‐ and ATP‐induced cytokine production by human blood‐derived immune cells (PBMCs and whole blood) was inhibited by silent agonists. Taken together, our results suggest that nAChRs modulate pro‐inflammatory monocyte numbers and function via receptor desensitization rather than channel opening. nAChR silent agonists may thus be a novel source of anti‐inflammatory compounds that could be used for the treatment of inflammatory disorders. Support or Funding Information This work was supported by the MS Society of Canada, the New Brunswick Health Research Foundation and NIH. This abstract is from the Experimental Biology 2019 Meeting. There is no full text article associated with this abstract published in The FASEB Journal .

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.007

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.008
GPT teacher head0.245
Teacher spread0.237 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2019
Admission routes3
Has abstractyes

Explore more

Same venueThe FASEB JournalSame topicNicotinic Acetylcholine Receptors StudyFrench-language works237,207