MétaCan
Menu
← Back to cohort
Record W3201337313 · doi:10.21203/rs.3.rs-31108/v1

A novel data-driven approach reveals gene networks and biological processes underlying autism

2020· preprint· en· W3201337313 on OpenAlexfundno aff
Leonardo Emberti Gialloreti, Roberto Enea, Valentina Di Micco, Daniele Di Giovanni, Paolo Curatolo

Bibliographic record

VenueResearch Square (Research Square) · 2020
Typepreprint
Languageen
FieldNeuroscience
TopicAutism Spectrum Disorder Research
Canadian institutionsnot available
FundersUniversity of British Columbia
KeywordsAutismComputational biologyGeneBiologyEvolutionary biologyNeuroscienceCognitive scienceGeneticsComputer scienceData sciencePsychologyDevelopmental psychology

Abstract

fetched live from OpenAlex

Abstract Background: Developments in gene-hunting techniques identified several ASD associated genes. The considerable significance of cluster analysis associated with gene network studies has led to reveal many disrupted key pathways in ASD, even if its genetic underpinnings remain a challenging task. This study aims to determine, through a novel data-driven approach, how networks of mutated genes impact biological processes underlying autism. Methods: We analyzed the VariCarta dataset, which presents more than 200,000 genomic variant events collected from 13,069 people with ASD. Firstly, we created a whole-genome and an exome sequencing subset. Then, for each subset we compared pairwise patients of each group to build “patient similarity matrices”. Hierarchical-agglomerative-clustering and heatmap were performed to identify clusters of patients with common occurrences of gene networks within these matrices. The subsequent enrichment analysis (EA) highlighted biological processes that might be impacted by the mutated genes of each subgroup. Results: Considering the whole-genome matrix, we identified three main genetic clusters of ASD patients, each one characterized by a network of shared genetic variants. We isolated 11,609 genetic variants shared by at least two subjects in each cluster; 4,187 of these variants (36.1%) were common to the three clusters. Only 331 patients (2.5%) shared none or very few mutated genes with anyone else. The EA highlighted common or cluster-specific biological processes related to the variants. Most of the common abnormal processes were involved in neuron projections guidance and morphogenesis, cell junctions and synapse assembly. Exome sequencing alone was not effectual in identifying ASD subgroups. Limitations: Caution is warranted when interpreting our results, as we did not compare them with a control group and did not verify if the identified subgroups where actually associated with different phenotypes. Future work will have to ascertain the strength and reproducibility of these results. Conclusions: Itemizing not just single mutated genes, but also gene networks and specific biological processes that characterize different ASD subpopulations might allow to better understand which networks of genetic variants play a major role in the etiopathology of ASD. The proposed methodology may represent a novel approach to help disentangle ASD complexity and an instrument to boost more focused genotype-phenotype studies.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.004
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Simulation or modeling · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: none
Teacher disagreement score0.005
Threshold uncertainty score0.009

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.004
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.002
Bibliometrics0.0030.002
Science and technology studies0.0010.000
Scholarly communication0.0010.001
Open science0.0010.001
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.505
GPT teacher head0.474
Teacher spread0.031 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designSimulation or modeling
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2020
Admission routes1
Has abstractyes

Explore more

Same venueResearch Square (Research Square)→Same topicAutism Spectrum Disorder Research→French-language works237,207→