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Record W4200577883 · doi:10.26685/urncst.295

The Use of the λ Bacteriophage to Neutralize the Pathogenic Effects of Shiga Toxins from Escherichia Coli to Combat Antimicrobial Resistance: A Research Protocol

2021· article· en· W4200577883 on OpenAlexaff
Jessica M. Karlovcec, Emma Veres, Brendan D. Paget

Bibliographic record

VenueUndergraduate Research in Natural and Clinical Science and Technology (URNCST) Journal · 2021
Typearticle
Languageen
FieldEnvironmental Science
TopicBacteriophages and microbial interactions
Canadian institutionsUniversity of Guelph
Fundersnot available
KeywordsEscherichia coliMicrobiologyLytic cycleShiga toxinBiologyAntibiotic resistanceAntimicrobialBacteriophageBacteriaPathogenic Escherichia coliPathogenic bacteriaReceptorAntibioticsVirologyGeneVirusGenetics

Abstract

fetched live from OpenAlex

Introduction: Resistance to antibiotics is becoming a global health crisis. Certain strains of Escherichia coli bacteria are resistant to antibiotic drugs and thrive without competition. Pathogenic effects of E. coli are caused by Shiga toxins they produce. Instead of attempting to kill the pathogens, we propose altering the bacterial genome of E. coli, causing the expression of globotriaosylceramide (Gb3) receptors, effectively neutralizing produced toxins. Methods: Phages are introduced to cultured E. coli cells and incorporate their DNA into the bacterial genome. Mutation vectors are transformed into E. coli cells using electroporation, causing expression of Gb3 receptors and preventing the lysing of the cell. Next, exposure to UV light causes phages to enter the lytic cycle, and mutated phages can be collected. Then, E. coli cells will be administered to 28 Wistar rats, and phage treatments causing expression of Gb3 receptors on E. coli cells will be administered to the treatment group. Rats in both groups will be monitored for symptomatology of E. coli poisoning, and stool samples will be collected and analyzed for quantities of Shiga toxins. Anticipated Results: We anticipate that phage-treated E. coli cells will express Gb3 receptors. We expect that, in the control group, symptoms of E. coli poisoning and quantities of Shiga toxins will increase over the duration of the study. Upon adequate expression of Gb3 receptors, we expect symptoms of E. coli poisoning and quantities of Shiga toxins to be lower in the treatment group than in the control group throughout the study. Discussion: Confirmation of our anticipated results through immunofluorescence spectra visualizing Gb3 receptors, histogram plots of symptoms, and lateral flow assays detecting quantities of Shiga toxins will prove our methods to be a valuable asset for decreasing the effects of E. coli poisoning and related diseases. Conclusion: Antibiotic resistance is becoming a serious threat. As antibiotic drugs are designed to kill bacteria, we propose an alternative method to neutralizing the pathogenic effects caused by E. coli through the incorporation of Gb3 receptors, and allowing the proliferation of the bacteria. This suggests an approach to slowing the acceleration of antibiotic resistance.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.002
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Protocol · Consensus signal: none
Teacher disagreement score0.009
Threshold uncertainty score0.031

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0020.001
Meta-epidemiology (narrow)0.0020.001
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0010.001
Science and technology studies0.0010.001
Scholarly communication0.0010.001
Open science0.0010.001
Research integrity0.0010.002
Insufficient payload (model declined to judge)0.0090.005

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.057
GPT teacher head0.391
Teacher spread0.334 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreProtocol

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2021
Admission routes1
Has abstractyes

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