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Record W4205310056 · doi:10.3410/f.1158602.618802

Faculty Opinions recommendation of Bcl-2 proteins EGL-1 and CED-9 do not regulate mitochondrial fission or fusion in Caenorhabditis elegans.

2009· dataset· en· W4205310056 on OpenAlexfundno aff
Sharad Kumar

Bibliographic record

VenueFaculty Opinions – Post-Publication Peer Review of the Biomedical Literature · 2009
Typedataset
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicMitochondrial Function and Pathology
Canadian institutionsnot available
FundersCanadian Institutes of Health ResearchNational Institutes of HealthUniversity of FloridaBurroughs Wellcome Fund
KeywordsMitochondrial fissionCaenorhabditis elegansmitochondrial fusionMitochondrionCell biologyBiologyFusion proteinMutantApoptosisMitochondrial DNAFissionGeneGenetics

Abstract

fetched live from OpenAlex

The Bcl-2 family proteins are critical apoptosis regulators that associate with mitochondria and control the activation of caspases.Recently, both mammalian and C. elegans Bcl-2 proteins have been implicated in controlling mitochondrial fusion and fission processes in both living and apoptotic cells.To better understand the potential roles of Bcl-2 family proteins in regulating mitochondrial dynamics, we carried out a detailed analysis of mitochondria in animals that either lose or have increased activity of egl-1 and ced-9, two Bcl-2 family genes that induce and inhibit apoptosis in C. elegans, respectively.Unexpectedly, we found that loss of egl-1 or ced-9, or overexpression of their gene products, had no apparent effect on mitochondrial connectivity or mitochondrial size.Moreover, loss of ced-9 did not affect the mitochondrial morphology observed in a drp-1 mutant, where mitochondrial fusion occurs but mitochondrial fission is defective, or in a fzo-1 mutant, where mitochondrial fission occurs but mitochondrial fusion is restricted, suggesting that ced-9 is not required for either the mitochondrial fission or fusion process in C. elegans.Taken together, our results argue against an evolutionarily conserved role for Bcl-2 proteins in regulating mitochondrial fission and fusion. RESULTS Mitochondrial morphogenesis is not affected in egl-1(lf) or ced-9(lf) mutantsRecently, the C. elegans pro-apoptotic BH3-only Bcl-2 protein EGL-1 has been implicated in promoting mitochondria fission during apoptosis [1].In addition, the C. elegans anti-apoptotic Bcl-2 protein CED-9 was shown to mediate mitochondria fission during apoptosis in one study [1] but was found to promote mitochondria fusion in healthy cells in another [2], calling into question of the exact physiological roles of C. elegans Bcl-2 family proteins in regulating mitochondria dynamics.To address the critical issue of whether Bcl-2 proteins regulate normal mitochondrial fission or fusion process in C. elegans, we carried out a comprehensive analysis of mitochondria morphology and structure in animals that either lose or have increased activity of egl-1 or ced-9.First, we visualized mitochondria in early C. elegans embryos that were

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.006
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Not applicable · Consensus signal: Not applicable
GenreCandidate signal: Dataset · Consensus signal: Dataset
Teacher disagreement score0.022
Threshold uncertainty score0.075

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.006
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0030.004
Science and technology studies0.0000.000
Scholarly communication0.0010.001
Open science0.0010.001
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0220.012

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.022
GPT teacher head0.326
Teacher spread0.305 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designNot applicable
Domainnot available
GenreDataset

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2009
Admission routes1
Has abstractyes

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