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Record W4221115800 · doi:10.1101/2022.03.18.484874

Per-Pathogen Virulence of HIV-1 subtypes A, C and D

2022· preprint· en· W4221115800 on OpenAlexaff
Judith A. Bouman, Colin M. Venner, Courtney J. Walker, Eric J. Arts, Roland R. Regoes

Bibliographic record

VenuebioRxiv (Cold Spring Harbor Laboratory) · 2022
Typepreprint
Languageen
FieldImmunology and Microbiology
TopicHIV Research and Treatment
Canadian institutionsWestern University
Fundersnot available
KeywordsVirulencePathogenViral loadBiologyVirologyHuman immunodeficiency virus (HIV)ImmunologyCd4 t cellSet pointLentivirusViral diseaseT cellGeneticsImmune systemGene

Abstract

fetched live from OpenAlex

Abstract HIV-1 subtypes differ, among other things, in their clinical manifestations and the speed in which they spread. In particular, the frequency of subtype C is increasing relative to subtype A and D. We aim to investigate whether HIV-1 subtype A, C and D differ in their per-pathogen virulence and to what extend this can explain the difference in spread between these subtypes. We use data from the Hormonal Contraception and HIV-1 Genital Shedding and Disease Progression among Women with Primary HIV Infection (GS) Study. For each study participant, we determine the set-point viral load value, CD4 + T cell level after primary infection and CD4 + T cell decline. Based on both the CD4 + T cell count after primary infection and CD4 + T cell decline, we estimate the time until AIDS for each individual. We then obtain our newly introduced measure of virulence as the inverse of the estimated time until AIDS. This new measure of virulence has an improved correlation with the set-point viral load compared to the decline of CD4 + T cells alone. After fitting a model to the measured virulence and set-point viral load values, we tested if this relation varies per subtype. We found that subtype C has a significantly higher per-pathogen virulence than subtype A. Based on an evolutionary model, we then hypothesize that differences in the primary length of infection period cause the observed variation in the speed of spread of the subtypes. Author summary HIV-1 subtype C is currently spreading relatively fast in various parts of the world. Data from a study that followed many women infected with different HIV-1 subtypes (A, C and D) before they started treatment shows that neither their viral load nor the disease duration are increased for subtype C compared to subtype A and D. Thus, it seems that subtype C does not have a transmission advantage, neither per contact nor due to longer infection, making the observed relative rise in subtype C a puzzle. We used the same data to test if subtype C has optimized its potential to spread by decreasing the disease duration per unit of viral load (per-pathogen virulence) compared to subtype A and D. However, we find that subtype C has a significantly higher per-pathogen virulence than subtype A and D. This result makes the rise of subtype C even more counter-intuitive. In a last step, we develop an evolutionary model, in which we synthesize all our empirical results. With this model we can show that the most likely explanation for the global spread of subtype C is a difference in the duration of primary infection between the subtypes.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.004
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.006
Threshold uncertainty score0.012

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.004
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.001
Bibliometrics0.0010.001
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0010.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0030.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.013
GPT teacher head0.229
Teacher spread0.216 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2022
Admission routes1
Has abstractyes

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