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Record W4231126669 · doi:10.1002/dvdy.24054

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2014· paratext· en· W4231126669 on OpenAlexafffund
Namal Abeysundara, Albert C. Leung, David A. Primrose, Sarah C. Hughes

Bibliographic record

VenueDevelopmental Dynamics · 2014
Typeparatext
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicHippo pathway signaling and YAP/TAZ
Canadian institutionsUniversity of Alberta
FundersInstitute of GeneticsInstitute of Zoology, Chinese Academy of SciencesRIKENFeinberg School of MedicineUniversity of California, San FranciscoUniversity of California, Santa BarbaraNational Institutes of HealthTokyo Metropolitan UniversityTechnion-Israel Institute of TechnologyUniversity of TokushimaGenome Institute of SingaporeWakayama Medical UniversityChinese Academy of SciencesUniversity of QueenslandMonash UniversityNational Cancer InstituteUniversity of BristolSchool of MedicineNewcastle UniversityUniversity of DundeeUniversity of South FloridaUniversity of OxfordBrigham Young UniversityUniversity College LondonCancer Research UKWellcome TrustNational Tsing Hua UniversityMontana State UniversityUniversity of Illinois at Urbana-ChampaignUniversity of DaytonMassachusetts General HospitalUniversity of MinnesotaCase Western Reserve UniversityUniversity of ArizonaNorthwestern UniversityUniversity of MiamiStowers Institute for Medical ResearchUniversity of South CarolinaCincinnati Children's Hospital Medical CenterBrown UniversityHarvard UniversityUniversity of WashingtonUniversity of Nebraska Medical CenterUniversity of TorontoYale University
KeywordsMerlin (protein)BiologyCell biologySuppressorScaffold proteinNeurofibromatosis type 2Signal transductionGeneticsNeurofibromatosisGene

Abstract

fetched live from OpenAlex

Background: The tumor suppressor protein merlin is thought to regulate cell proliferation and cell adhesion through interaction with protein partners. Loss of merlin is associated with Neurofibromatosis Type 2 (NF2) tumors. NHERF1 or EBP50 is a scaffolding protein that functions in apical organization of polarized cells. Merlin and NHERF1 have been shown to interact in vitro in vertebrates. We investigate how the Drosophila NHERF1 orthologue, Sip1, and Merlin function to regulate cell proliferation and adhesion. Results: We identify two conserved arginine residues (R325 and R335) in Merlin which, in addition to the FERM domain, are required for interaction with Sip1. Mutation of the arginine residues result in reduced Sip1 binding to Merlin and loss of Merlin growth suppressor function. Over‐expression of Merlin R325A and/or Merlin R335L in Drosophila wings result in increased proliferation in the adult wing (increase in size), which is rescued by co‐over‐expression of constitutively active Merlin protein. Reduced Sip1 binding to Merlin also produces defects in adhesion in follicle epithelial cells. Conclusions: Sip1 facilitates the activation of Merlin as a tumor suppressor protein. Thus, our work provides insight into how Merlin functions as a tumor suppressor and in adhesion and this provides insight into the mechanism of NF2 pathogenesis. Developmental Dynamics 243:1554–1570, 2014 . © 2014 Wiley Periodicals, Inc.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow), Insufficient payload (model declined to judge)
Consensus categoriesInsufficient payload (model declined to judge)
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Not applicable · Consensus signal: Not applicable
GenreCandidate signal: Other · Consensus signal: Other
Teacher disagreement score0.226
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0010.000
Insufficient payload (model declined to judge)0.0010.020

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.006
GPT teacher head0.225
Teacher spread0.220 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; both teacher heads agree on what is shown here.

Study designNot applicable
Domainnot available
GenreOther

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2014
Admission routes2
Has abstractyes

Explore more

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