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Record W4239497743 · doi:10.25011/cim.v30i4.2881

Regulation of huntingtin palmitoylation and its role in Huntington Diseases

2007· article· en· W4239497743 on OpenAlexvenueaboutno aff
F BJ Young, M R Hayden

Bibliographic record

VenueClinical and investigative medicine · 2007
Typearticle
Languageen
FieldNeuroscience
TopicGenetic Neurodegenerative Diseases
Canadian institutionsnot available
Fundersnot available
KeywordsPalmitoylationHuntingtinPolyglutamine tractHuntingtin ProteinCell biologyHuntington's diseaseBiologyMutationMutantNeuroscienceGeneticsCysteineBiochemistryGeneInternal medicineMedicine

Abstract

fetched live from OpenAlex

Huntington’s Disease (HD) is an autosomal dominant neurodegenerative disorder characterized by motor, cognitive, and psychiatric deficits and selective neuronal cell death. The causative mutation in HD is an expansion of the N-terminal polyglutamine tract in huntingtin (htt), which results in altered trafficking of mutant htt and enhanced toxicity to striatal neurons. Post-translational modification by the lipid palmitate has been shown to play a critical role in the trafficking and function of many proteins, including htt. It has been previously demonstrated that huntingtin-interacting protein 14 (HIP14) is a palmitoyl transferase that palmitoylates htt. Previous characterization of HIP14 demonstrated a reduced interaction with mutant htt resulting in reduced palmitoylation, suggesting that palmitoylation may play a role in the pathogenesis of HD. Most recently, we have identified cysteine 214 as a major site of htt palmitoylation in the N-terminus of htt, close to the site of polyglutamine expansion. It was demonstrated that mutation of this site, rendering htt palmitoylation-resistant, results in increased neuronal toxicity, enhanced inclusion formation, and in altered trafficking of htt. Remarkably, mutation of the palmitoylation site in wild type htt also resulted in enhanced toxicity similar to that seen in mutant htt. Together, these previous studies suggest a critical role of palmitoylation in htt trafficking and function. Based on this preliminary work, we are characterizing the enzymatic regulation of huntingtin palmitoylation. Exploring htt palmitoylation in a number of existing and new mouse models imparts key insights into how this process is regulated in vivo. We are also exploring the relationship between palmitoylation and other post-translational modifications of htt. These studies will lead to an understanding of the regulation of palmitoylation of huntingtin in vivo, as well as setting the precedent to understand the general role of palmitoylation in a wide range of other human diseases. Ultimately, this may lead to identification of new therapeutic targets and treatments for patients. F.B.J.Y. is supported by a Canadian Institutes of Health Research Walter and Jessie Boyd & Charles Scriver - Child and Family Research Institute - UBC MD/PhD Studentship Award. She also receives funding from the Michael Smith Foundation for Health Research as a Junior Trainee.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.004

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.117
GPT teacher head0.346
Teacher spread0.229 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2007
Admission routes2
Has abstractyes

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