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Abstract 16139: Mast Cells Promote Proliferation and Suppress Myogenic Differentiation of Mesenchymal Stem Cells

2015· article· en· W4251217398 on OpenAlexaff
Mansoreh Nazari, Nathan C. Ni, Ana Lüdke, Shuhong Li, Jian Guo, Terrence M. Yau, Richard D. Weisel, Ren‐Ke Li

Bibliographic record

VenueCirculation · 2015
Typearticle
Languageen
FieldMedicine
TopicTissue Engineering and Regenerative Medicine
Canadian institutionsToronto General HospitalUniversity of Toronto
Fundersnot available
KeywordsMyocardinMesenchymal stem cellMyofibroblastCellular differentiationKLF4MedicineCell biologyCell growthEndocrinologyCancer researchInternal medicineBiologyPathologyEmbryonic stem cellSerum response factorFibrosisSOX2

Abstract

fetched live from OpenAlex

Introduction: Efficient scar formation post-myocardial infarction (MI) depends on fibroblast/mesenchymal stem cell (MSC) accumulation and myofibroblast transdifferentiation. Mast cell (MC)-deficient mice exhibit reduced cardiac myofibroblast accumulation and undergo rapid left ventricular dilation post-MI. Thus, we sought to investigate how MCs modulate MSC proliferation and their myogenic differentiation into myofibroblasts. Methods: MC/MSC co-culture was used to examine MC effects on MSC proliferation and differentiation. MC granulate (MCG) was then used to further examine the mechanisms by which MCs exerted their effects on MSC proliferation, migration, and myogenic differentiation. The effects of MCs on MSC activity in vivo was evaluated by immunohistological analysis on MC- and saline-treated infarcted mouse hearts at 3 and 7 days post-MI Results: MC co-culture suppressed expression of the myogenic differentiation marker α-smooth muscle actin in MSCs. Similarly, MCG dose-dependently decreased myogenic differentiation by up to ~90% (P<0.01, n=4) and increased MSC proliferation ~two-fold (P<0.01, n=5) and migration by ~75% (P<0.01, n=4). Pharmacological antagonism of platelet-derived growth factor receptor (PDGFR) rescued MSC differentiation despite MCG treatment, suggesting MC suppression of MSC differentiation occurs through PDGFR. MCG treatment resulted in up to ~70% decreased miR-145 and -143 expression (P<0.05, n=4), as well as increased Klf4 (~25%) and decreased myocardin (~50%) protein expression in MSCs, indicating that the myocardin-Klf4 axis mediates MSC proliferation/differentiation. Finally, infarcted hearts from mice treated with MCs showed ~five-fold increased CD29+ MSC proliferation at day 3 vs saline-treated animals (P<0.05, n=3). This effect was absent by day 7 post-MI. Conclusions: MSCs can oscillate between proliferative and differentiated states. MCs promote proliferation at the expense of differentiation early after MI through PDGFR, downregulation of miR-145 and -143, and the Klf4-myocardin signalling axis to promote MSC accumulation. This in turn results in a larger cardiac MSC pool for later myofibroblast differentiation, facilitating improved wound healing.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.008
Threshold uncertainty score0.028

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0080.002

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.028
GPT teacher head0.248
Teacher spread0.220 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2015
Admission routes1
Has abstractyes

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