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Record W4295088693 · doi:10.1093/brain/awac330

<i>ATP6V0C</i> variants impair V-ATPase function causing a neurodevelopmental disorder often associated with epilepsy

2022· article· en· W4295088693 on OpenAlexaff
Kari A. Mattison, Gilles Tossing, Fred Mulroe, Callum Simmons, Kameryn M. Butler, Alison Schreiber, Adnan Alsadah, Derek Neilson, K Naess, Anna Wedell, Anna Wredenberg, Arthur Sorlin, Emma McCann, George J. Burghel, Beatriz Menéndez, George Hoganson, Lorenzo D. Botto, Francis Filloux, Ángel Aledo‐Serrano, António Gil‐Nagel, Katrina Tatton‐Brown, Nienke E. Verbeek, Bert van der Zwaag, Kyrieckos A. Aleck, Andrew C. Fazenbaker, Jorune Balciuniene, Holly Dubbs, Eric D. Marsh, Kathryn B. Garber, Jakob Ek, Morten Dunø, Christina Engel Hoei‐Hansen, Matthew A. Deardorff, Gordana Raca, Catherine Quindipan, Michèle Van Hirtum-Das, Jeroen Breckpot, Trine Bjørg Hammer, Rikke S. Møller, Andrea Whitney, Andrew G. L. Douglas, Mira Kharbanda, Nicola Brunetti‐Pierri, Manuela Morleo, Vincenzo Nigro, Halie May, James X. Tao, Emanuela Argilli, Elliot H. Sherr, William B. Dobyns, Richard A. Baines, Jim Warwicker, Jennifer Parker, Siddharth Banka, Andrew Escayg

Bibliographic record

VenueBrain · 2022
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicATP Synthase and ATPases Research
Canadian institutionsUniversité de Montréal
FundersNational Eye InstituteMedical Research CouncilNational Heart, Lung, and Blood InstituteWinship Cancer InstituteCitizens United for Research in EpilepsyFonds Wetenschappelijk OnderzoekDepartment of Health and Social CareNational Institute of Neurological Disorders and StrokeNational Institute of General Medical SciencesNational Institute for Health and Care ResearchNational Institutes of HealthCancer Research UKVlaamse regeringEuropean CommissionBroad InstituteNational Human Genome Research InstituteWellcome TrustNational Cancer InstituteEmory University
KeywordsEpilepsyNeuroscienceNeurodevelopmental disorderMedicinePsychologyPsychiatryAutism

Abstract

fetched live from OpenAlex

The vacuolar H+-ATPase is an enzymatic complex that functions in an ATP-dependent manner to pump protons across membranes and acidify organelles, thereby creating the proton/pH gradient required for membrane trafficking by several different types of transporters. We describe heterozygous point variants in ATP6V0C, encoding the c-subunit in the membrane bound integral domain of the vacuolar H+-ATPase, in 27 patients with neurodevelopmental abnormalities with or without epilepsy. Corpus callosum hypoplasia and cardiac abnormalities were also present in some patients. In silico modelling suggested that the patient variants interfere with the interactions between the ATP6V0C and ATP6V0A subunits during ATP hydrolysis. Consistent with decreased vacuolar H+-ATPase activity, functional analyses conducted in Saccharomyces cerevisiae revealed reduced LysoSensor fluorescence and reduced growth in media containing varying concentrations of CaCl2. Knockdown of ATP6V0C in Drosophila resulted in increased duration of seizure-like behaviour, and the expression of selected patient variants in Caenorhabditis elegans led to reduced growth, motor dysfunction and reduced lifespan. In summary, this study establishes ATP6V0C as an important disease gene, describes the clinical features of the associated neurodevelopmental disorder and provides insight into disease mechanisms.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.830
Threshold uncertainty score0.596

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.009
GPT teacher head0.239
Teacher spread0.230 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations32
Published2022
Admission routes1
Has abstractyes

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