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Cellular Senescence Is Immunogenic and Promotes Antitumor Immunity

2022· letter· en· 325 citations· W4307493936 on OpenAlex· 10.1158/2159-8290.cd-22-0523

Why is this work in the frame?

A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.

Canadian affiliationAn author listed a Canadian institution. This is the only route the usual frame has.
Canadian funderA Canadian agency funded it. The work may carry no Canadian affiliation at all.

Full frame distilled prediction

Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

Candidate categories
Meta-epidemiology (narrow), Research integrity, Insufficient payload (model declined to judge)
Consensus categories
none
Domain
Candidate signal: noneConsensus signal: none
Study design
Candidate signal: Not applicableConsensus signal: Not applicable
Genre
Candidate signal: CommentaryConsensus signal: none
Teacher disagreement score
0.390
Threshold uncertainty score
1.000
Validation status
machine_predicted_unvalidated · codex-gemma-dda1882f352a

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.001
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.003
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Opus teacher head0.022
GPT teacher head0.263
Teacher spread
0.241 · how far apart the two teachers sit on this one work
Validation status
score_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Abstract

Cellular senescence is a stress response that activates innate immune cells, but little is known about its interplay with the adaptive immune system. Here, we show that senescent cells combine several features that render them highly efficient in activating dendritic cells (DC) and antigen-specific CD8 T cells. This includes the release of alarmins, activation of IFN signaling, enhanced MHC class I machinery, and presentation of senescence-associated self-peptides that can activate CD8 T cells. In the context of cancer, immunization with senescent cancer cells elicits strong antitumor protection mediated by DCs and CD8 T cells. Interestingly, this protection is superior to immunization with cancer cells undergoing immunogenic cell death. Finally, the induction of senescence in human primary cancer cells also augments their ability to activate autologous antigen-specific tumor-infiltrating CD8 lymphocytes. Our study indicates that senescent cancer cells can be exploited to develop efficient and protective CD8-dependent antitumor immune responses. SIGNIFICANCE: Our study shows that senescent cells are endowed with a high immunogenic potential-superior to the gold standard of immunogenic cell death. We harness these properties of senescent cells to trigger efficient and protective CD8-dependent antitumor immune responses. See related article by Chen et al., p. 432. This article is highlighted in the In This Issue feature, p. 247.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

The record

Venue
Cancer Discovery
Topic
Telomeres, Telomerase, and Senescence
Field
Medicine
Canadian institutions
Université de MontréalCentre Hospitalier Universitaire Sainte-Justine
Funders
Departament d'Empresa i Coneixement, Generalitat de CatalunyaInstituto de Salud Carlos IIINatural Sciences and Engineering Research Council of CanadaFondation Charles-BruneauCentre de recherche du CHU Sainte-JustineFonds de Recherche du Québec - SantéHarald och Greta Jeanssons StiftelseBanco Bilbao Vizcaya ArgentariaVetenskapsrådetFundación Científica Asociación Española Contra el CáncerMinisterio de Ciencia e InnovaciónGeneralitat de CatalunyaCancerfondenAgència de Gestió d'Ajuts Universitaris i de RecercaCanadian Institutes of Health ResearchFundación Bancaria Caixa d'Estalvis i Pensions de BarcelonaFundación BBVAKarolinska InstitutetLoo och Hans Ostermans Stiftelse för Medicinsk ForskningInstitute for Research in BiomedicineInstitució Catalana de Recerca i Estudis AvançatsCanada Foundation for Innovation
Keywords
Immune systemSenescenceCD8Cytotoxic T cellBiologyAntigen presentationAcquired immune systemCancer cellMHC class ICell biologyAntigenContext (archaeology)Cross-presentationImmunogenic cell deathImmunologyT cellCancerImmunotherapyIn vitro
Has abstract in OpenAlex
yes