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Record W4307493936 · doi:10.1158/2159-8290.cd-22-0523

Cellular Senescence Is Immunogenic and Promotes Antitumor Immunity

2022· letter· en· W4307493936 on OpenAlexafffund
Inés Marín, Olga Boix, Andrea García-Garijo, Isabelle Sirois, Adrià Caballé, Eduardo Zarzuela, Irene Ruano, Camille Stephan‐Otto Attolini, Neus Prats, José A López-Domínguez, Marta Kovatcheva, Elena Garralda, Javier Muñoz, Étienne Caron, María Abad, Alena Gros, Federico Pietrocola, Manuel Serrano

Bibliographic record

VenueCancer Discovery · 2022
Typeletter
Languageen
FieldMedicine
TopicTelomeres, Telomerase, and Senescence
Canadian institutionsUniversité de MontréalCentre Hospitalier Universitaire Sainte-Justine
FundersDepartament d'Empresa i Coneixement, Generalitat de CatalunyaInstituto de Salud Carlos IIINatural Sciences and Engineering Research Council of CanadaFondation Charles-BruneauCentre de recherche du CHU Sainte-JustineFonds de Recherche du Québec - SantéHarald och Greta Jeanssons StiftelseBanco Bilbao Vizcaya ArgentariaVetenskapsrådetFundación Científica Asociación Española Contra el CáncerMinisterio de Ciencia e InnovaciónGeneralitat de CatalunyaCancerfondenAgència de Gestió d'Ajuts Universitaris i de RecercaCanadian Institutes of Health ResearchFundación Bancaria Caixa d'Estalvis i Pensions de BarcelonaFundación BBVAKarolinska InstitutetLoo och Hans Ostermans Stiftelse för Medicinsk ForskningInstitute for Research in BiomedicineInstitució Catalana de Recerca i Estudis AvançatsCanada Foundation for Innovation
KeywordsImmune systemSenescenceCD8Cytotoxic T cellBiologyAntigen presentationAcquired immune systemCancer cellMHC class ICell biologyAntigenContext (archaeology)Cross-presentationImmunogenic cell deathImmunologyT cellCancerImmunotherapyIn vitro

Abstract

fetched live from OpenAlex

Cellular senescence is a stress response that activates innate immune cells, but little is known about its interplay with the adaptive immune system. Here, we show that senescent cells combine several features that render them highly efficient in activating dendritic cells (DC) and antigen-specific CD8 T cells. This includes the release of alarmins, activation of IFN signaling, enhanced MHC class I machinery, and presentation of senescence-associated self-peptides that can activate CD8 T cells. In the context of cancer, immunization with senescent cancer cells elicits strong antitumor protection mediated by DCs and CD8 T cells. Interestingly, this protection is superior to immunization with cancer cells undergoing immunogenic cell death. Finally, the induction of senescence in human primary cancer cells also augments their ability to activate autologous antigen-specific tumor-infiltrating CD8 lymphocytes. Our study indicates that senescent cancer cells can be exploited to develop efficient and protective CD8-dependent antitumor immune responses. SIGNIFICANCE: Our study shows that senescent cells are endowed with a high immunogenic potential-superior to the gold standard of immunogenic cell death. We harness these properties of senescent cells to trigger efficient and protective CD8-dependent antitumor immune responses. See related article by Chen et al., p. 432. This article is highlighted in the In This Issue feature, p. 247.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Not applicable · Consensus signal: Not applicable
GenreCandidate signal: Commentary · Consensus signal: Commentary
Teacher disagreement score0.004
Threshold uncertainty score0.007

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.001
Scholarly communication0.0010.000
Open science0.0000.000
Research integrity0.0040.002
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.022
GPT teacher head0.263
Teacher spread0.241 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designNot applicable
Domainnot available
GenreCommentary

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations325
Published2022
Admission routes2
Has abstractyes

Explore more

Same venueCancer DiscoverySame topicTelomeres, Telomerase, and SenescenceFrench-language works237,207