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Record W4362606390 · doi:10.1038/s41586-023-05874-3

Lung adenocarcinoma promotion by air pollutants

2023· article· en· W4362606390 on OpenAlex

Why this work is in the frame

A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.

affAt least one author lists a Canadian institution in the pinned OpenAlex snapshot.
fundA Canadian funder is recorded on the work.

Bibliographic record

VenueNature · 2023
Typearticle
Languageen
FieldEnvironmental Science
TopicAir Quality and Health Impacts
Canadian institutionsOntario Institute for Cancer ResearchVancouver Coastal HealthVancouver Coastal Health Research InstituteUniversity of British Columbia
FundersLinda Crnic Institute for Down Syndrome, University of Colorado School of Medicine, Anschutz Medical CampusNational Cancer InstituteJapan Society for the Promotion of ScienceEngineering and Physical Sciences Research CouncilHorizon 2020 Framework ProgrammeGenentechPeking University People's HospitalOno PharmaceuticalUniversity College London Hospitals NHS Foundation TrustChinese Academy of Medical SciencesMedical Research CouncilNational Research Foundation of KoreaCancerfondenVetenskapsrådetNational Natural Science Foundation of ChinaPeking UniversityChang Gung Medical FoundationNational Institute for Health and Care ResearchAstraZenecaInvitaeRevolution MedicinesAstex PharmaceuticalsLifeArcFrancis Crick InstituteWellcome TrustCancer Research UKUniversity College LondonStand Up To CancerMinistry of Science and ICT, South KoreaLUNGevity FoundationEntertainment Industry FoundationBristol-Myers SquibbEuropean Respiratory SocietyPfizerNational Institutes of HealthRosetrees TrustEuropean CommissionBreast Cancer Research FoundationNational Research FoundationAmgenAmerican Association for Cancer ResearchGénome QuébecKorea Health Industry Development InstituteInternational Association for the Study of Lung CancerU.S. Department of Veterans Affairs
KeywordsLung cancerKRASCarcinogenesisCancer researchLungAdenocarcinomaMedicineCarcinogenCancerImmunologyOncologyBiologyInternal medicineGeneticsColorectal cancer

Abstract

fetched live from OpenAlex

A complete understanding of how exposure to environmental substances promotes cancer formation is lacking. More than 70 years ago, tumorigenesis was proposed to occur in a two-step process: an initiating step that induces mutations in healthy cells, followed by a promoter step that triggers cancer development1. Here we propose that environmental particulate matter measuring ≤2.5 μm (PM2.5), known to be associated with lung cancer risk, promotes lung cancer by acting on cells that harbour pre-existing oncogenic mutations in healthy lung tissue. Focusing on EGFR-driven lung cancer, which is more common in never-smokers or light smokers, we found a significant association between PM2.5 levels and the incidence of lung cancer for 32,957 EGFR-driven lung cancer cases in four within-country cohorts. Functional mouse models revealed that air pollutants cause an influx of macrophages into the lung and release of interleukin-1β. This process results in a progenitor-like cell state within EGFR mutant lung alveolar type II epithelial cells that fuels tumorigenesis. Ultradeep mutational profiling of histologically normal lung tissue from 295 individuals across 3 clinical cohorts revealed oncogenic EGFR and KRAS driver mutations in 18% and 53% of healthy tissue samples, respectively. These findings collectively support a tumour-promoting role for PM2.5 air pollutants and provide impetus for public health policy initiatives to address air pollution to reduce disease burden. Combination of epidemiology, preclinical models and ultradeep DNA profiling of clinical cohorts unpicks the inflammatory mechanism by which air pollution promotes lung cancer

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesInsufficient payload (model declined to judge)
Consensus categoriesInsufficient payload (model declined to judge)
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Not applicable · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.903
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0010.002

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.020
GPT teacher head0.310
Teacher spread0.290 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it