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Sequential and interwoven requirements for HEB and Id3 in fetal gamma-delta T cell commitment and functional programming

2023· article· en· W4385686279 on OpenAlexaff
Michele K. Anderson, Johanna Selvaratnam, Juliana Dutra Barbosa da Rocha, Vinothkumar Rajan, Helen Wang, Emily C. Reddy, Cynthia J. Guidos, Juan Carlos Zúñiga‐Pflücker

Bibliographic record

VenueThe Journal of Immunology · 2023
Typearticle
Languageen
FieldImmunology and Microbiology
TopicImmune Cell Function and Interaction
Canadian institutionsHospital for Sick ChildrenSunnybrook HospitalUniversity of TorontoSickKids FoundationSunnybrook Health Science Centre
Fundersnot available
KeywordsT-cell receptorBiologyT cellTranscription factorFetusCell biologyCytotoxic T cellImmunologyMolecular biologyGeneGeneticsPregnancyImmune system

Abstract

fetched live from OpenAlex

Abstract Two major developmental steps are necessary for the generation of innate IL-17 producing γδ T cells. First, T cell precursors in the fetal thymus commit to either the αβ or the γδ T cell lineage. This is followed by a second stage of differentiation into subsets that can produce either IL-17 (γδT17) or IFNγ (γδT1) cells. Both developmental events are dependent on TCR signal strength, but how this is interpreted at the molecular level has been unclear. Id3, which antagonizes the activity of HEB transcription factors, is upregulated in proportion to TCR signal strength, providing an important link between TCR signaling and gene expression. We previously found that γδ T cells in mice lacking HEB were impaired in IL-17 production. To better understand the molecular basis of this defect, we conducted single cell RNA-sequencing on fetal thymic γδ T cells. In WT cells, the ratio of HEB to Id3 was highest in populations undergoing γδ T cell commitment, whereas later populations were dominated by Id3. Comparing scRNA-seq datasets from WT and HEB-deficient γδ T cells revealed a profound shift in TCRγ and TCRδ chain expression. This was accompanied by a decrease in key regulators of early γδ T cell development. Id3 expression was also severely decreased. Accordingly, Id3-deficient mice were impaired in their ability to generate γδT17 cells. However, γδ T cells from Id3-deficient mice showed a decrease in second stage γδT17 regulators, rather than the first stage regulators defective in HEB-deficient mice. Therefore, distinct ratios of HEB and Id3 are required during the γδ T cell commitment, and HEB is instrumental in inducing Id3 to enable functional programming of γδT17 cells in the fetal thymus. Supported by grants from CIHR (201610PJT), NSERC (RGPIN-2020-05596) and NIH (1P01AI102853-06)

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.007

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0000.001
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.032
GPT teacher head0.271
Teacher spread0.238 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2023
Admission routes1
Has abstractyes

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