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Record W4387008461 · doi:10.1101/2023.09.24.23295485

Common and Rare Variant Contributions to Bradyarrhythmias from Multi-Ancestry Meta-Analyses

2023· preprint· en· W4387008461 on OpenAlexaff
Lu‐Chen Weng, Joel Rämö, Sean J. Jurgens, Shaan Khurshid, Mark Chaffin, Amelia Weber Hall, Valerie N. Morrill, Victor Nauffal, Yan V. Sun, Dominik Beer, Simon Lee, Girish N. Nadkarni, ThuyVy Duong, Biqi Wang, Tomasz Czuba, Thomas R. Austin, Zachary T. Yoneda, Daniel J. Friedman, Anne Clayton, Matthew C. Hyman, Renae Judy, Allan C. Skanes, Kate M. Orland, Timothy Treu, Matthew T. Oetjens, Álvaro Alonso, Elsayed Z. Soliman, Honghuang Lin, Kathryn L. Lunetta, Jesper van der Pals, Tariq Z. Issa, Navid A. Nafissi, Heidi T. May, Peter Leong‐Sit, Carolina Roselli, Seung Hoan Choi, Habib Khan, Stacey Knight, Richard Karlsson Linnér, Connie R. Bezzina, Samuli Ripatti, J. Michael Gaziano, Ruth J. F. Loos, Bruce M. Psaty, J. G. Smith, Emelia J. Benjamin, Dan E. Arking, Daniel J. Rader, Svati H. Shah, Dan M. Roden, Scott M. Damrauer, Lee L. Eckhardt, Jason D. Roberts, Michael J. Cutler, M. Benjamin Shoemaker, Christopher M. Haggerty, Kelly Cho, Aarno Palotie, Peter W.F. Wilson, Patrick T. Ellinor, Steven A. Lubitz

Bibliographic record

VenuemedRxiv · 2023
Typepreprint
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicGenetic Associations and Epidemiology
Canadian institutionsWestern University
FundersCommon FundNational Institute on Drug AbuseNational Human Genome Research InstituteNational Institute of Neurological Disorders and StrokeNational Institute of Mental HealthNIH Office of the DirectorNational Heart, Lung, and Blood InstituteNational Cancer InstituteNational Institutes of Health
KeywordsGeneticsHeritabilityGenome-wide association studyBiologyLMNAExome sequencingExomeSick sinus syndromeCandidate geneMissing heritability problemInternal medicineBioinformaticsGenotypeMedicineGenePhenotypeGenetic variantsSingle-nucleotide polymorphism

Abstract

fetched live from OpenAlex

ABSTRACT To broaden our understanding of bradyarrhythmias and diseases of the cardiac conduction system, we performed cross-sectional multi-ancestry genome-wide association study meta-analyses in up to 1.3 million individuals for sinus node dysfunction (SND), distal conduction disease (DCD), and pacemaker implantation (PM). We evaluated the biological relevance of bradyarrhythmia loci by analyses of transcriptomes, pleiotropy, and partitioned heritability based on cardiac single cell RNA sequencing data. Finally, we performed rare variant burden testing in 460,000 whole exome sequenced individuals from two biobanks. We identified 13, 28, and 21 common variant loci for SND, DCD, and PM, respectively. Four well-known common variant arrhythmia loci ( SCN5A/SCN10A , CCDC141, TBX20 , and CAMK2D) were shared for SND and DCD, while other loci were more specific for either SND or DCD. Cardiomyocyte-expressed genes were strongly enriched for contributions to DCD heritability, while SND and PM were more heterogeneous. Rare variant analyses implicated LMNA for all bradyarrhythmia subtypes; SMAD6 and SCN5A for DCD; and TTN , MYBPC3 , and SCN5A for PM. The genetic architectures of SND and DCD are both overlapping and distinct. Multiple genetic mechanisms involving ion channels, sarcomeric components, cellular homeostasis, and cardiac development may influence the development of bradyarrhythmias.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.015
metaresearch head score (Gemma)0.021
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Meta-analysis · Consensus signal: Meta-analysis
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.015
Threshold uncertainty score0.077

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0150.021
Meta-epidemiology (narrow)0.0020.001
Meta-epidemiology (broad)0.0040.022
Bibliometrics0.0030.005
Science and technology studies0.0010.001
Scholarly communication0.0030.001
Open science0.0020.002
Research integrity0.0020.002
Insufficient payload (model declined to judge)0.0040.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.169
GPT teacher head0.406
Teacher spread0.237 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designMeta-analysis
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2023
Admission routes1
Has abstractyes

Explore more

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