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Record W4387009599 · doi:10.21203/rs.3.rs-3304066/v1

The causality between telomere length and chronic lung diseases: A Bidirectional Mendelian Randomization Analysis

2023· preprint· en· W4387009599 on OpenAlexaff
Yuan Zhan, Yiya Gu, Ruonan Yang, Zhesong Deng, Shanshan Chen, Qian Huang, Jixing Wu, Jinkun Chen, Jungang Xie

Bibliographic record

VenueResearch Square · 2023
Typepreprint
Languageen
FieldMedicine
TopicTelomeres, Telomerase, and Senescence
Canadian institutionsWestern University
FundersNational Natural Science Foundation of China
KeywordsMendelian randomizationSingle-nucleotide polymorphismLung cancerGenome-wide association studyInternal medicineMedicineGastroenterologyOncologyBiologyGeneticsGenotypeGene

Abstract

fetched live from OpenAlex

Abstract Background Previous studies have observed the abnormality in telomere biology and function during the process of chronic lung diseases (CLDs). However, whether alteration of telomere length (TL) causally facilitates the incidence of CLDs remains to be determined. Therefore, we here aim to estimate the causal effect of TL on the risk of CLDs using mendelian randomization (MR) analysis. Methods Single nucleotide polymorphisms (SNPs) strongly associated with TL and CLDs were selected as genetic variables from the genome-wide association studies (GWAS). A bidirectional two-sample MR analysis primarily based on inverse variance weighted (IVW) method was then conducted to infer the causality between TL and CLDs. Cochran’s Q test and MR-Egger regression analysis were performed to assess the heterogeneity and pleiotropy, and leave-one-out analysis was tested to determine the stability of MR results. Results The forward MR analysis indicated that among non-neoplastic CLDs, elevated TL was causally related to reduced risk of asthma (OR = 0.9986, 95%CI 0.9972–0.9999, P = 0.035), chronic obstructive pulmonary disease (COPD) (OR = 0.9987, 95%CI 0.9975–0.9999, P = 0.040), idiopathic pulmonary fibrosis (IPF) (OR = 0.9971, 95%CI 0.9961–0.9980, P < 0.001), and sarcoidosis (OR = 0.6820, 95%CI 0.5236–0.8884, P = 0.005). For neoplastic CLDs, increased TL genetically predicted higher risk of non-small cell lung cancer (OR = 1.8485, 95%CI 1.4074–2.4279, P < 0.001) and lung adenocarcinoma (OR = 1.9636, 95%CI 1.2275–3.1412, P = 0.005). However, there presented no significant causality between TL and pulmonary arterial hypertension, pneumoconiosis, small cell lung cancer and squamous cell lung cancer. Moreover, reverse MR analysis all showed no obvious causalities of CLDs with TL, except for sarcoidosis (OR = 0.9936, 95%CI 0.9887–0.9984, P = 0.010). Sensitivity analyses suggested the robustness of MR results with no horizonal pleiotropy despite of partial heterogeneity in reverse MR analysis. Conclusions Our study demonstrates that TL is causally associated with decreased risk of several non-neoplastic CLDs (asthma, COPD and IPF), whereas associated with increased risk of non-small cell lung cancer (especially adenocarcinoma). There’s mutual causality between TL attrition and sarcoidosis onset. This study comprehensively elucidated the causal associations between TL and CLDs, and might provide a promising target for the prevention of these CLDs.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.049
metaresearch head score (Gemma)0.072
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.049
Threshold uncertainty score0.260

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0490.072
Meta-epidemiology (narrow)0.0020.001
Meta-epidemiology (broad)0.0030.007
Bibliometrics0.0020.002
Science and technology studies0.0010.001
Scholarly communication0.0010.001
Open science0.0020.002
Research integrity0.0020.002
Insufficient payload (model declined to judge)0.0080.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.075
GPT teacher head0.419
Teacher spread0.344 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2023
Admission routes1
Has abstractyes

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