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P32 Multiple Ammonia-Induced Episodes of Hepatic Encephalopathy Provoke Neuronal Cell Loss in Bile-Duct Ligated Rats

2023· article· en· W4387041330 on OpenAlexaff
Farzaneh Tamnanloo, Rafael Ochoa‐Sanchez, Mariana M. Oliveira, Mélanie Tremblay, Christopher F. Rose

Bibliographic record

VenueThe American Journal of Gastroenterology · 2023
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicMitochondrial Function and Pathology
Canadian institutionsUniversité de Montréal
Fundersnot available
KeywordsMedicineHepatic encephalopathyHyperammonemiaOxidative stressHippocampusInternal medicineEndocrinologyEncephalopathyHippocampal formationBrain damagePathologyAnesthesia

Abstract

fetched live from OpenAlex

Background: Hepatic encephalopathy (HE) is a major complication of chronic liver disease (CLD). This neurological disorder is known to be reversible following liver transplantation (LT). Retrospective studies have demonstrated an association between a history of HE episodes and poor neurological outcome following LT. However, the impact of HE episodes on the brain remains unknown. Our aim was to evaluate the impact of multiple episodes on neurological status and brain injury in rats with CLD. Method: Five-week BDL rats and Sham-operated controls were used. BDL rats were injected with ammonium acetate, precipitating an overt episode of HE every 4 days from week 3 post-surgery (total; 4 episodes). Sham rats also received ammonia and control rats received saline. Three days after the last episode, short- and long-term memory (LTM) were assessed. Upon sacrifice, plasma and brains were collected for oxidative stress measurements and western blot used to investigate neuronal integrity in frontal cortex, hippocampus, and cerebellum. Proteomic analysis was performed on hippocampal tissue samples. Results: LTM was found to be impaired in both BDL-Vehicle and BDL-Ammonia groups vs respective Sham. However, LTM impairment was further aggravated in BDL-Ammonia rats. In BDL-Ammonia, GFAP and apoptotic markers (cleaved caspase-3 and Bax/Bcl2) were increased in the hippocampus, whereas neuronal markers (NeuN and SMI311) were reduced compared to all other experimental groups. Plasma oxidative stress was increased in BDL-Ammonia compared to respective Sham and BDL rats. Decreased total antioxidant capacity and increased 4-HNE levels were found in the hippocampus of the BDL-Ammonia group compared to respective sham and BDL rats. These differences observed in the multiple episodic BDL rats were not demonstrated following one episode. Proteomic analysis revealed 2 proteins which significantly altered in BDL-Ammonia compared to BDL-Vehicle and respective Sham. Conclusion: Multiple episodes of overt HE exacerbated the neurological impairments in BDL rats. LTM impairment in the BDL-Ammonia rats was associated with increased oxidative stress, apoptotic markers and decreased neuronal markers in the hippocampus, suggesting neuronal injury/loss. Elevated levels of GFAP in the hippocampus insinuate gliosis. These results suggest that cumulative HE episodes may cause permanent cell damage and, therefore, will less likely reversible following LT.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.008

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0010.000
Science and technology studies0.0000.001
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.011
GPT teacher head0.240
Teacher spread0.229 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2023
Admission routes1
Has abstractyes

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