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Modulation of the BK Channel During Acute Spinal Cord Injury is Neuroprotective

2016· article· en· W4389007752 on OpenAlexafffundabout
Marianne Jacobsen, Kristin A Lett, Max Karnitsky, Balsam M Arwini, John M. Barden, Josef Buttigieg

Bibliographic record

VenueThe FASEB Journal · 2016
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicIon channel regulation and function
Canadian institutionsUniversity of Regina
FundersSaskatchewan Health Research Foundation
KeywordsNeuroprotectionSpinal cord injuryMedicineSpinal cordExcitotoxicityAnesthesiaNeurosciencePharmacologyInternal medicineBiologyReceptor

Abstract

fetched live from OpenAlex

Spinal cord injury results in significant neuronal and glial cell death resulting in impaired neurological and motor function. While there are often surviving axonal tracts, these are frequently dysmyelinated or demyelinated. Key to this pathology is uncontrolled Ca 2+ entry, resulting in excitotoxicity and cell death. We propose that by activating mechanisms attenuating global depolarization and limiting Ca 2+ entry, we can reduce neuronal death and significantly improve neurological and motor function. To this effect, we previously demonstrated that the large‐conductance, voltage and Ca 2+ ‐activated K + (BK or Maxi K + ) channel is altered after SCI, and may play a part in the neuropathology of SCI. In this study, we set out to explore whether the use of a BK channel activator, Isopimaric acid (ISO), is neuroprotective post spinal cord injury. Wister rats received a 25‐g clip compression injury at the T7 levels for 1 min. One hour post‐surgery, they were administered ISO (120 μM), Ibeurotoxin (IbTx; 100nM), or a vehicle control via mini‐osmotic pump. Complete gait analysis was performed using two‐dimensional motion capture by filming rats on a weekly basis for 8 weeks. Compound Action Potentials (CAP) and high frequency conduction experiments were performed on spinal cords 8 weeks post surgery. Finally, the size of the injury site, loss of myelination, and cell death were assessed using immunohistochemistry (IHC). Complete gait analysis suggests that treatment with ISO improves motor function in our test model. ISO treated animals were able to recover gait function to a greater extent than IbTx and control animals. While the IbTx treated rats also demonstrated an improvement in complete gait function, their recovery was delayed and not of the same magnitude as ISO treated animals. This finding was further supported in our electrophysiology and IHC experiments. In our CAP experiments we find that there is a significant difference between all three groups. Additionally, we found preservation or re‐establishment of axonal tracts and myelination in our IHC experiments. In conclusion, activation of the BK channel during acute SCI may be a novel therapeutic target for acute spinal cord injury. Further studies are required to determine if modulation of the BK channel in chronic injury is beneficial for recovery, and to understand the mechanisms by which this ion channel may be working during spinal cord injury repair. Support or Funding Information Saskatchewan Health Research Foundation

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.005

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.014
GPT teacher head0.252
Teacher spread0.238 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2016
Admission routes3
Has abstractyes

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