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PDE1C regulates the dynamics of actin‐based structures in migrating human arterial smooth muscle cells

2017· article· en· W4389018089 on OpenAlexaffabout
Paulina Brzezińska, Darrin Payne, Sarah Rampersad, Jodi Mackeil, Jonah Burke‐Kleinman, Donald H. Maurice

Bibliographic record

VenueThe FASEB Journal · 2017
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicPhosphodiesterase function and regulation
Canadian institutionsQueen's University
Fundersnot available
KeywordsForskolinCell biologyPseudopodiaChemistryGene knockdownCell migrationCyclic adenosine monophosphateActinPhosphodiesteraseCellBiologyBiochemistryReceptorApoptosisEnzyme

Abstract

fetched live from OpenAlex

Phenotypic remodeling and an associated increased migration by human arterial smooth muscle cells (HASMCs) are features of neo‐intimal hyperplasia that promote occlusive vascular diseases, including post‐angioplasty restenosis. Although it is established that cyclic adenosine 3′, 5′‐monophosphate (cAMP)‐mediated signaling inhibits overall HASMC migration, how this ubiquitous signaling system impacts the individual “steps” required for directed cell migration, including the dynamics of lamellipodial projections at their leading edges, is currently poorly defined. Using a modification of the trans‐well assay, our study highlights a critical role for a specific cyclic nucleotide phosphodiesterase, Phosphodiesterase 1C (PDE1C), in modulating lamellipodial dynamics in migrating HASMCs. Formation of HASMC lamellipodial projections in response to exposure of these cells to a chemotactic gradient (0.5% FBS) was assessed by quantifying the accumulation of actin‐rich structures on the underside of filters with 3 μm pores in 4hrs. When compared to vehicle‐treated controls, selective pharmacological inhibition of PDE1C (compound 33 (C‐33), Dart Neuroscience LLC) increased accumulation of lamellipodial structures significantly. Similarly, RNAi‐mediated knockdown of PDE1C using several distinct siRNA markedly increased accumulation of lamellipodial structures when compared to those formed in control siRNA transfected cells. In contrast, agents that increase intracellular cAMP by activating HASMC adenylyl cyclases (ACs) (i.e. forskolin) reduced accumulation of these actin‐rich structures in HASMCs exposed to a gradient of FBS. Most interestingly, lamellipodial projection accumulation in response to PDE1C inhibition, or knockdown, was also markedly antagonized by addition of forskolin. These results are consistent with the novel idea that PDE1C is acting locally at the leading edge of HASMCs to promote accumulation of lamellipodial structures and that this hyper‐localized effect is lost when HASMC cAMP levels are increased globally, using forskolin. Consistent with this, we show that PDE1C can associate with an A‐kinase anchoring protein AKAP79 and the cAMP effector Protein Kinase A (PKA). Overall, since each PDE1C and AKAP79 is found at the leading edge of migrating HASMCs, we suggest that PDE1C acts locally, via a PKA/AKAP79‐based signalosome, to regulate lamellipodial projection formation in migrating HASMCs. Support or Funding Information Canadian Institutes for Health Research (CIHR)

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.097
Threshold uncertainty score0.448

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0010.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.012
GPT teacher head0.253
Teacher spread0.241 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations2
Published2017
Admission routes2
Has abstractyes

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