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The chemosensory bitter taste receptors (T2Rs) are involved in proliferation and migration of breast cancer

2017· article· en· W4389021533 on OpenAlexaffabout
Nisha Singh, Feroz Ahmed Shaik, Rajinder P. Bhullar, Prashen Chelikani

Bibliographic record

VenueThe FASEB Journal · 2017
Typearticle
Languageen
FieldNursing
TopicBiochemical Analysis and Sensing Techniques
Canadian institutionsUniversity of Manitoba
Fundersnot available
KeywordsCell growthBreast cancerCancer cellCancer researchCell migrationSKBR3ReceptorApoptosisBiologySignal transductionCell biologyCellCancerChemistryBiochemistryHuman breast

Abstract

fetched live from OpenAlex

Background and Objective Breast cancer is a very complex disease and involves interactions between many proteins. The human bitter taste receptors (T2Rs) are a group of 25 chemosensory proteins that belong to the G protein‐coupled receptor family. T2Rs mediate signal transduction in response to stimulation by a wide variety of bitter compounds. Earlier studies showed that bitter agonists such as quinidine and chloroquine triggered apoptosis in MCF‐7 breast cancer cells, and bitter melon extract inhibited breast cancer cell proliferation by modulating cell cycle regulatory genes and promoting apoptosis. However, the cell surface receptor (T2R) targets for these bitter compounds, and their expression and characterization in the cancer cells is not yet elucidated. Methods To delineate the expression profile of T2Rs in breast cancer MDA‐MB‐231 and MCF‐7 cells and non‐cancerous MCF‐10A cell line, quantitative PCR (qPCR) and flow cytometry analysis was done. Next, analysis of T2R4 and T2R49 in human breast cancer tissue panel was performed using qPCR array (Origene Inc.). Calcium mobilization experiment was pursued to analyze the functional response after stimulation of these cells using bitter compounds. Cell proliferation was pursued using MTT assay and migration analysis by RTCA xCELLigence cell system after stimulating the cells with T2R ligands. Results Our results suggest differential expression of T2Rs is breast cancer cells. Stimulation of endogenous T2Rs with bitter agonists leads to increase in intracellular calcium release. Breast cancer cells treated with T2R4 agonist show decrease in cell proliferation and migration compared to non‐cancerous cells. This effect was reversed upon T2R4 antagonist treatment. Furthermore, knockdown and/or overexpression studies of T2R4 in these cells suggest its role in inhibition of cell proliferation and migration of breast cancer cells. Conclusions Our results suggest that T2Rs are involved in decrease of breast cancer cell proliferation and migration. Investigating the mechanistic role of these chemosensory T2Rs in breast cancer would aid in developing strategies to prevent and/or treat breast cancer. Support or Funding Information The Natural Sciences and Engineering Research Council of Canada (NSERC)

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.004
Threshold uncertainty score0.013

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0000.001
Bibliometrics0.0000.001
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0040.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.020
GPT teacher head0.266
Teacher spread0.247 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2017
Admission routes2
Has abstractyes

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