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The Effect of Cardiolipin on Microglial Activation and Microglia‐Mediated Neuronal Death

2017· article· en· W4389023524 on OpenAlexaff
Caitlin B. Pointer, Andis Klegeris

Bibliographic record

VenueThe FASEB Journal · 2017
Typearticle
Languageen
FieldNeuroscience
TopicNeuroinflammation and Neurodegeneration Mechanisms
Canadian institutionsOkanagan University CollegeUniversity of British Columbia, Okanagan CampusUniversity of British Columbia
Fundersnot available
KeywordsMicrogliaNeuroinflammationCell biologyChemistryReactive oxygen speciesExtracellularInflammationBiologyImmunology

Abstract

fetched live from OpenAlex

Microglia, the immune cells of the brain, become activated in response to a variety of stimuli. Under normal physiological conditions, microglial activation is advantageous, as it produces an acute inflammatory response that helps rid the brain of the noxious stimuli through the release of pro‐inflammatory mediators and cytotoxins, including reactive oxygen species (ROS) and reactive nitrogen species (RNS). When microglial activation is sustained, however, the persistent and non‐specific release of these cytotoxic molecules results in damage to surrounding cells, including neurons and other microglia. Such state of chronic neuroinflammation is a characteristic feature of Alzheimer's disease (AD). Cardiolipin (CL), a phospholipid located in the inner mitochondrial membrane, is significantly reduced in AD brains. The remaining CL is exposed to the high levels of ROS present in AD brains, which modifies the structure of CL and hinders its ability to regulate mitochondrial processes. This results in the further production of ROS and extensive neuron death. The intracellular role of CL is well defined; however, the effects of extracellular CL on microglial activation, and subsequently on neuronal viability, have not been described. We utilized cell culture techniques to study the effects of extracellular CL on the activation of microglia, and their subsequent toxicity towards neurons. BV‐2 murine microglia, as well as human HL‐60 promyelocytic and THP‐1 monocytic cells (microglia models), were exposed to CL in the presence of noxious stimuli for 24 or 48 hours. Pro‐inflammatory mediators including tumor necrosis factor (TNF)‐alpha, monocyte chemoattractant protein (MCP)‐1, ROS and RNS in supernatant were quantified to determine the effect CL has on the secretory profile of activated microglia. Additionally, THP‐1 cell supernatants were transferred onto human SH‐SY5Y neuroblastoma cells (neuron model) to determine whether CL regulates microglia‐mediated neurotoxicity. We discovered that CL decreased the secretion of TNF‐alpha, MCP‐1, ROS and RNS from activated microglial cells. Additionally, CL reduced the cytotoxicity of activated THP‐1 cells towards SH‐SY5Y neuronal cells. These findings indicate that CL may act as a novel intercellular signaling molecule that is capable of reducing microglial activation, as well as neuron death. Further studies will explore glial signaling pathways engaged by CL and reveal its role in neuron‐microglia communication.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.003

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.025
GPT teacher head0.265
Teacher spread0.240 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2017
Admission routes1
Has abstractyes

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