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<i>Entamoeba histolytica</i> Stimulates the Release of the Alarmin Molecule HMGB1 upon Contact with Macrophages

2016· article· en· W4389024135 on OpenAlexafffund
Sharmin Begum, Joëlle St‐Pierre, France Moreau, Kris Chadee

Bibliographic record

VenueThe FASEB Journal · 2016
Typearticle
Languageen
FieldMedicine
TopicAmoebic Infections and Treatments
Canadian institutionsUniversity of Calgary
FundersNatural Sciences and Engineering Research Council of Canada
KeywordsHMGB1SecretionEntamoeba histolyticaBiologyInflammasomeCell biologyMicrobiologyInflammationExtracellularImmune systemHistoneImmunologyBiochemistry

Abstract

fetched live from OpenAlex

Entamoeba histolytica ( Eh ) is the causative agent of amebiasis, which occurs in 10% of infected individuals when the parasite invades the underlying colonic mucosa. Parasite‐induced host pro‐inflammatory responses are critical in disease pathogenesis, but the mechanisms underlying this response is poorly illustrated. The contact between Eh and macrophages triggers a raging pro‐inflammatory response by activating NLRP3 inflammasome (IL‐1β) and other uncharacterized pathways (TNF‐α). High Mobility Group Box 1 protein (HMGB1) is a critical mediator of inflammation during infection and it shapes the magnitude and degree of the inflammatory response. HMGB1 is a non‐histone nuclear protein, which have overlapping binding side with histone H1 in the chromatin. It is released in the extracellular space during infection by activated or damaged immune cells and act as an alarmin molecule. In this study, we investigated the early release of HMGB1 upon Eh infection with macrophages and identification of the parasite virulent factors that initiated this response. Human monocytic cell line (THP‐1) and mouse bone marrow derived macrophages (BMDMs) were treated with live Eh , Eh components for different time point and western blotting was used to quantify HMGB1 secretion. Only stimulation with live Eh but not Eh components triggered robust time‐dependent (as early as 5 minutes) secretion of HMGB1. HMGB1 release from macrophage was contact‐dependent as inhibiting the Gallectin adhesion with exogenous galactose abrogated HMGB1 secretion. Another Eh surface virulent factor cysteine protease 5 ( Eh CP5) partially inhibited HMGB1 release, and the enzymatic activity of the CP5 was not required for the secretion. Moreover, HMGB1 protein secretion was independent of caspase‐1 activation, as secretion was not inhibited with the Pan caspase inhibitor, Z‐VAD‐fmk. Similar results were obtained using BMDMs from caspase‐1 − / − mice treated with live Eh . Surprisingly, there was the simultaneous release of the nuclear protein histone H1 along with the HMGB1 upon Eh infection. These results demonstrate that the potent pro‐inflammatory mediator HMGB1 release is the earliest innate response upon Eh contact with macrophages. This mediator can act by itself or along with other molecules as a danger signal (alarmin response) to sense invasive infection that can attract bystander cells and/or promote extensive inflammation, which is critical in disease pathogenesis. Support or Funding Information Grant Support: NSERC

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.006

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.008
GPT teacher head0.232
Teacher spread0.224 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2016
Admission routes2
Has abstractyes

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