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O0105 CROHN’S DISEASE AS AN INNATE IMMUNE DEFECT —THE CRITICAL ROLE OF CARD15 IN INTESTINAL ANTIBACTERIAL DEFENSE

2004· article· en· W4390567845 on OpenAlexaff
Bernadette Bègue, C. Dumant, Jean–Christophe Bambou, Jean‐François Beaulieu, Marco Giovannini, Nadine Cerf–Bensussan, Dana J. Philpott, Frank M. Ruemmele

Bibliographic record

VenueJournal of Pediatric Gastroenterology and Nutrition · 2004
Typearticle
Languageen
FieldImmunology and Microbiology
TopicImmunodeficiency and Autoimmune Disorders
Canadian institutionsUniversité de Sherbrooke
Fundersnot available
KeywordsMedicineImmunology

Abstract

fetched live from OpenAlex

Introduction: Recently, with the discovery of different mutations in the CARD15 gene in a subgroup of patients with ileal Crohn’s disease, a first clue to the understanding of the molecular mechanisms of this disorder was found. CARD15 is an intracel-lular protein functioning as a bacterial sensor via its leucine rich repeat recognizing bacterial muramyl-dipeptide, the minimal signature found in peptidoglycan of gram−/+ or /− bacteria. Methods: Intestinal epithelial cells (HIEC, Caco-2, HT-29) were stimulated by various pro-inflammatory cytokines and the induction of CARD4/CARD15 was monitored by semi-quantiative and quantitative real-time PCR. The functionality of CARD15 was tested by intracellular micro-injection of MDP or MDP-fugene-transfection by means of the activation of NFkB (EMSA, immunofluorescence). WT-CARD15 and mutated CARD15 (3020InsC) were transfected (electroporation method) into HIEC and Caco-2 cells and the response to MDP activation was measured using an NFkB reporter assay. The generation of a pro-inflammatory and anti-bacterial response to MDP-stimulation was compared between WT and mutant transfected enterocytes using a real time PCR approach. Results: Normal, non-transformed intestinal epithelial cells clearly expressed CARD4, but no CARD15 signal was observed. However, pro-inflammatory stimuli induced a high degree of CARD15 expression in an NFkB dependent (TNF) or –independent (IFN) manner. Cytokine-induced CARD15 in enterocytes was functional, since after stimulation with MDP a marked activation of NFkB pathway was observed, as demonstrated by the nuclear translocation of the p65 subunit and by EMSA. Cells not expressing CARD15 were unresponsive to MDP. Using an NFkB reporter assay, we observed that enterocytes transfected with a mutant form of CARD15 failed to activate NFkB. Whereas WT transfectants generated a marked pro-inflammatory response with enhanced anti-bacterial defence activation in response to MDP, mutants were not able to generate either a pro-inflammatory or anti-bacterial (defensin) response. Conclusion: There is clear experimental evidence that in CD patients with a CARD15 mutation, the inducible innate immune system on the intestinal epithelial cell level is defective. In the present study, we observed that in response to bacterial products, normal enterocytes generate a pro-inflammatory and an extremely potent immediate anti-bacterial response. This antibacterial ability is clearly impaired in CD patients rendering them more susceptible to elicit an inappropriate inflammatory reaction to various bacterial stimuli.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.480
Threshold uncertainty score0.545

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0010.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.005
GPT teacher head0.230
Teacher spread0.225 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2004
Admission routes1
Has abstractyes

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