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Record W4391873584 · doi:10.1093/jcag/gwad061.045

A45 ASSESSING THE ROLE OF MICROBIOTA DYSBIOSIS ON INTESTINAL TUFT CELL FUNCTIONS UPON <i>GIARDIA</i> INFECTION

2024· article· en· W4391873584 on OpenAlexaff
Olivia Sosnowski, Thibault Allain, Derek M. McKay, André G. Buret

Bibliographic record

VenueJournal of the Canadian Association of Gastroenterology · 2024
Typearticle
Languageen
FieldNursing
TopicNutrition, Health and Food Behavior
Canadian institutionsUniversity of Calgary
Fundersnot available
KeywordsDysbiosisTuftGiardiaBiologyMicrobiologyImmunologyGut flora

Abstract

fetched live from OpenAlex

Abstract Background Enteric tuft cells can detect and respond to intestinal parasitic infection by modulating host responses to aid parasite clearance, yet the extent of tuft cell functions upon other infections remains to be elucidated. Previous work in our lab showed that the enteric protozoan parasite Giardia induced tuft and goblet cell hyperplasia, yet Giardia colonization in Pou2f3-/- (tuft cell-deficient) mice was impaired. Not only can Giardia modulate the gut microbiota and lead to microbial dysbiosis, but the microbiota itself can influence Giardia colonization. Interestingly, certain microbiota-derived products can activate tuft cells in the gut. In this study, we investigated the role of microbiota dysbiosis in the crosstalk between Giardia and tuft cells. Aims We aim to explore the bidirectional effects between tuft cells and Giardia infection by investigating the role of 1) the host microbiota influenced by the presence or absence of tuft cells in altering Giardia infectivity, and 2) Giardia modified microbiota in contributing to tuft cell activity. Methods Fecal matter from 6–8-week-old C57Bl/6 and Pou2f3-/- mice were administered to polyethylene glycol (PEG) treated C57Bl/6 mice via oral gavage for 7 days, followed by gavage with 5x104Giardia muris trophozoites. Duodenal parasite burden was assessed 11 days after infection. Small intestinal (SI) microbiota was collected from 5–7-week-old C57Bl/6 and Pou2f3-/- mice following 11 days of G. muris infection and transplanted into PEG treated C57Bl/6 and Pou2f3-/- recipients. 11 days post-transfer, SI tuft and goblet cell hyperplasia was assessed in recipient mice by immunohistochemistry, and Dclk1, Atoh1, and Muc2 gene expression was quantified by RT-qPCR. Parameters of gut motility were evaluated. Results FMT with Pou2f3-/- mouse fecal matter prior to Giardia infection led to improved weight gain during infection compared to FMT from C57Bl/6 mice. Giardia modified microbiota (GMM) did not induce tuft or goblet cell hyperplasia 11 days after transfer. Jejunal Dclk1, Atoh1, and Muc2 were no different after GMM compared to control microbiota (CM) transfer. Ileal Atoh1 or Muc2 were upregulated in mice receiving GMM from C57Bl/6 or Pou2f3-/- mice, respectively. Intestinal transit time and stool water content was altered in mice which received GMM from Pou2f3-/- mice compared to CM. Conclusions The resident microbiota in tuft cell deficient mice may in part contribute to limiting Giardia infection. Giardia modified microbiota does not substantially contribute to tuft cell hyperplasia or related gene expression alterations at 11 days after microbiota transfer. The mechanistic contributors to tuft cell hyperplasia during Giardia infection and how a lack of tuft cells can lead to impaired colonization does not appear to be primarily driven by microbiota alterations and remains to be fully defined. Funding Agencies NSERC

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.070
Threshold uncertainty score0.942

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0010.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.008
GPT teacher head0.249
Teacher spread0.241 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2024
Admission routes1
Has abstractyes

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