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Record W4396991831 · doi:10.1681/asn.20213210s1409a

Rare Variants in Syndromic Ciliopathy Genes as Novel Causes of Isolated Renal Disease in Adults

2021· article· en· W4396991831 on OpenAlexaff
Zachary T. Sentell, Lina Mougharbel, Sima Babayeva, Natascia Anastasio, Lee Lee Chu, Murielle M. Akpa, Paul Goodyer, Elena Torban, Thomas M. Kitzler

Bibliographic record

VenueJournal of the American Society of Nephrology · 2021
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicGenetic and Kidney Cyst Diseases
Canadian institutionsMcGill University Health Centre
Fundersnot available
KeywordsCiliopathyDiseaseGeneMedicineNephrologyGeneticsBiologyInternal medicinePhenotype

Abstract

fetched live from OpenAlex

Background: Renal ciliopathies are among the commonest genetic causes of endstage renal disease (ESRD). Ciliopathies are caused by defects of the primary cilium, an antenna-like organelle with mechanosensory roles, crucial for organ development and maintenance. Disorders of the cilium present early with multi-organ involvement, but some individuals present as adults with organ-specific phenotypes, potentially due to milder mutations and organ-specific effects. Methods: We identified rare variants in two ciliopathy genes in two unrelated adults presenting with ESRD. ACMG guidelines did not classify these variants as pathogenic, requiring functional validation to establish a causal genotype-phenotype relationship. Results: Bi-allelic C2CD3 missense variants were identified in a proband with ESRD, suggestive of an isolated renal ciliopathy. C2CD3 is essential for ciliogenesis, with complete loss of cilia in knockout mice (Development 135:4049 2008). Severe mutations were reported in patients with a syndromic ciliopathy (OFD XIV; OMIM# 615948), but no cases of isolated renal disease have been reported. We detected a moderate but consistent shortened cilia length in skin fibroblasts and renal epithelial cells from our proband, suggestive of a milder ciliary defect. Remarkably, the proportion of ciliated cells was significantly reduced in renal epithelial cells but not in fibroblasts, indicating an organ-specific ciliogenesis defect. Pathogenic variants in CC2D2A cause Joubert and Meckel syndrome, with no isolated renal presentations observed to date (Mol. Genet. Genom. e1603 2021). We identified a novel homozygous nonsense variant (Arg34*) in CC2D2A, classified as not pathogenic due to an alternate start-codon, in a previously healthy 37-year-old male with isolated ESRD of unknown etiology. Using public data (GTEx), we show that protein-coding transcripts harbouring this variant are the predominant trancripts in the kidney when compared to tissues relevant to CC2D2A-related phenotypes (e.g., cerebellum, liver). Conclusions: Rare variants in known syndromic ciliopathy genes cause isolated renal disease in adults due to potential organ-specifc effects. Using variant classification schemes without functional analysis may not accurately capture the genetic contribution to adult ESRD. Funding: Government Support - Non-U.S.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.010

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0010.001
Science and technology studies0.0010.000
Scholarly communication0.0000.000
Open science0.0000.001
Research integrity0.0010.000
Insufficient payload (model declined to judge)0.0030.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.007
GPT teacher head0.238
Teacher spread0.231 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2021
Admission routes1
Has abstractyes

Explore more

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