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Record W4396993456 · doi:10.1681/asn.20213210s1235b

Constitutive Activation of Hedgehog Signaling in FOXD1+ Renal Stromal Cells Impairs Ureteric Epithelial Branching via CXCL12

2021· article· en· W4396993456 on OpenAlexaff
Robert D’Cruz, Yun‐Kyo Kim, Norman D. Rosenblum

Bibliographic record

VenueJournal of the American Society of Nephrology · 2021
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicRenal and related cancers
Canadian institutionsHospital for Sick Children
Fundersnot available
KeywordsStromal cellHedgehog signaling pathwayHedgehogUreteric budCancer researchCell biologyStromaChemistryInternal medicineEndocrinologyBiologyMedicineSignal transductionKidney developmentBiochemistryGeneImmunohistochemistry

Abstract

fetched live from OpenAlex

Background: Renal dysplasia, characterized by disorganization of stromal tissue, malformation of collecting ducts, and decreased nephron number, is a major cause of renal failure in children. Nephrons and collecting ducts develop via reciprocal inductive interactions between two intermediate mesodermal tissue elements - the Osr1+ metanephric mesenchyme (MM), within which reside Six2+ nephrogenic and Foxd1+ stromal cell progenitors, and the Hoxb7+ ureteric bud, which gives rise to ureteric branches and their daughter collecting ducts. The key roles of expanded Foxd1+ stromal progenitors in the genesis of dysplastic tissue are largely undefined. Here, we identify a novel pathogenic stromal Hh-Cxcl12 signaling axis driving impaired ureteric epithelial branching. Methods: Mice with deficiency of Ptch1 specific to FOXD1+ stromal cells (Foxd1Cre;Ptch1loxP/-; referred to as stromal Ptch1-deficient) were analyzed by immunostaining, single-cell RNA sequencing, and bulk RNA sequencing. To investigate Cxcl12 as a downstream target of increased stromal Hh signaling, in vivo analysis of ureteric branching was assessed in Cxcl12 compound mutants (Foxd1Cre; Ptch1loxP-; Cxcl12loxP/+). Results: Stromal Ptch1-deficient kidneys exhibited renal dysplasia characterized by a 41% reduction in nephron number (P<0.01, n=4) at E18.5 and a 26% reduction in ureteric epithelial branch tips at E12.5 (P<0.01, n=6). Whole kidney qRT-PCR analysis at E13.5 further demonstrated a reduction in ureteric branching effectors Gdnf, Wnt11, Etv4, and Etv5 in stromal Ptch1-deficient mice (P<0.05, n=4). Bulk RNA sequencing of stromal Ptch1-deficient whole kidneys revealed a significant upregulation in numerous medullary stromal markers including the secreted stromal cell-derived factor Cxcl12 (P<0.01, n=3). Complementary single-cell RNA sequencing analysis further demonstrated upregulation of Cxcl12 in medullary stromal clusters of Ptch1-deficient kidneys relative to control samples. Analysis of branching morphogenesis in compound mutant mice heterozygous for Cxcl12 in a stromal Ptch1-deficient background revealed a complete rescue of ureteric epithelial branching defects (P<0.001, n=6). Conclusions: A pathogenic increase in stromal Hh signaling in mice causes nephron deficiency and impaired ureteric epithelial branching due to increased Cxcl12 activation. Funding: Government Support - Non-U.S.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.006

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.006
GPT teacher head0.235
Teacher spread0.229 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2021
Admission routes1
Has abstractyes

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