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Record W4396994765 · doi:10.1681/asn.20213210s1517a

Podocytes Soften in Proteinuric CKD: A Potential New Mechanism for Proteinuria?

2021· article· en· W4396994765 on OpenAlexaff
Luisa Ulloa Severino, Xiaolin He, Franziska Lausecker, Rachel Lennon, Mira Krendel, Darren A. Yuen, Yuen Lab

Bibliographic record

VenueJournal of the American Society of Nephrology · 2021
Typearticle
Languageen
FieldMedicine
TopicRenal Diseases and Glomerulopathies
Canadian institutionsSt. Michael's Hospital
Fundersnot available
KeywordsProteinuriaMechanism (biology)UrologyMedicineInternal medicineKidney diseaseEndocrinologyKidneyPhysics

Abstract

fetched live from OpenAlex

Background: Proteinuria is one of the most common manifestations of glomerular injury, and an important predictor of disease progression. Podocytes are a critical component of the glomerular filtration barrier, and as such podocyte injury is a major cause of proteinuria. Historically, investigators have focused on biochemical changes in podocytes that occur following podocyte injury. In contrast, little is known about the changes in the biophysical properties of these highly specialized epithelial cells. Methods: 3 different models of proteinuric glomerular disease were studied: (1) Akita+/- Ren+/- mice (a murine model of diabetes and renin-mediated hypertension that we have recently shown develops progressive glomerulosclerosis that mimics human diabetic kidney disease, n = 7), (2) Myo1e-/- mice (a mouse model of genetic FSGS characterized by deficiency of a non-muscle myosin involved in actomyosin contraction, n = 6), and (3) Col4a5-/- mice, a mouse model of Alport's syndrome (n = 12). The stiffness of glomeruli, glomerular basement membrane (GBM), and podocytes was measured using atomic force microscopy (AFM) and associated histology (picrosirius red, silver, and WT1 staining). Similar stiffness measurements were performed in human FSGS and healthy kidney donor biopsies. Results: AFM measurements revealed that glomerular stiffness increased in Akita+/- Ren+/- and Col4a5-/- mice, a finding that correlated with the degree of glomerulosclerosis. Glomerular stiffness was not increased in Myo1e-/- mice. GBM stiffness was increased in Akita+/- Ren+/- and Myo1e-/- mice, but not in Col4a5-/- Alport's mice. In all 3 mouse models, as well as in human FSGS biopsies, podocytes were softer than in healthy control kidneys. Conclusions: Taken together, these are the first data demonstrating that podocytes soften in proteinuric glomerular disease. Given the major rearrangements in the actomyosin network that occur in podocytes of proteinuric kidneys, our data suggest that podocyte softening may be a common manifestation of podocyte injury. Further work is ongoing to understand whether this softening is solely a consequence of podocyte injury, or if it also contributes to ongoing podocyte damage.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: none
Teacher disagreement score0.003
Threshold uncertainty score0.010

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.001
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0010.000
Science and technology studies0.0000.002
Scholarly communication0.0010.002
Open science0.0010.001
Research integrity0.0030.002
Insufficient payload (model declined to judge)0.0030.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.013
GPT teacher head0.274
Teacher spread0.261 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2021
Admission routes1
Has abstractyes

Explore more

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