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Record W4397045802 · doi:10.1681/asn.20223311s11b

Leveraging AKI Recovery Patterns Reveals a Genome-Wide Significant Variant Associated With a Higher Risk of Non-Resolving AKI

2022· article· en· W4397045802 on OpenAlexaff
Pavan K. Bhatraju, Ian B. Stanaway, Rajasree Menon, Jennifer A. Schaub, Matthias Kretzler, T. Alp İkizler, Edward D. Siew, Vernon M. Chinchilli, Amit X. Garg, Steven G. Coca, Alan S. Go, James S. Kaufman, Paul L. Kimmel, Chirag R. Parikh, Mark M. Wurfel, Jonathan Himmelfarb

Bibliographic record

VenueJournal of the American Society of Nephrology · 2022
Typearticle
Languageen
FieldMedicine
TopicRenin-Angiotensin System Studies
Canadian institutionsWestern University
Fundersnot available
KeywordsMedicineIntensive care medicineAcute kidney injuryInternal medicineComputational biologyBiology

Abstract

fetched live from OpenAlex

Background: Genetic studies have focused on associations between genetic variants and the risk for AKI compared to controls, but this framework may be limited because AKI is highly heterogeneous. We hypothesized that a GWAS for distinct AKI recovery patterns (resolving versus non-resolving AKI) would identify novel and robust genetic risks for AKI. Methods: We included 2,271 patients with AKI previously enrolled in three different studies of hospitalized patients (ASSESS-AKI, iSPAAR and HMC Trauma). Resolving AKI was a ≥ 0.3 mg/dL or ≥ 25% decrease in serum creatinine within the first 72 hours after AKI onset (n=1524) and non-resolving AKI was the absence of resolving (n=747). Array-based genome-wide genotypes were obtained from each dataset and imputed with Haplotype Reference Consortium v r1.1. We pooled results from all three studies and used an additive genetic model adjusting for site, age, sex, and the first 10 principal components (significance was p<5x10-8). Results: We identified one variant (rs263152, C>T, frequency 29%) on chromosome 6 that achieved genome-wide significance with the T allele associated with a greater risk of non-resolving AKI (OR=1.47, 95% CI: 1.28-1.68, p=4.28x10-8) (Figure 1). rs263152 is intronic to LOC153910, a long non-coding RNA. In each cohort, the minor allele of rs263152 was consistently associated with a greater risk of non-resolving AKI. Query of the NephQTL database revealed that rs263152 is a cis eQTL for AIG1 (Androgen Induced Gene-1) in tubulointerstitial cells (p=0.007). AIG1 has been implicated in albuminuria in the Framingham Heart Study.Figure 1.: Genome wide significant variant is associated with non-resalving AKI compared to resolving AKI. A. Quantile-quantile plot showing overall adherence to expected p values. The genomic inflation factor based on a median chi-square was estimated at 0.997, indicating negligible variation in population structure between cases and controls. B. Manhattan plot demonstrates rs263152, an intronic SNP in chromosome 6, is significantly associated with a greater risk of non-resolving AKI (OR=1.47, 95% CI: 1.28-1.68, p=4.28x10 -8). C. Forest plot demonstrates a consistent direction of effect with the minor allele of rs263152 associated with a greater risk of non-resolving AKI in all three studies. The size of the boxes represents the sample size of each study.Conclusions: Identification of genetic risks for AKI may be facilitated by a focus on less heterogeneous sub-phenotypes, such as non-resolving AKI. Our findings suggest that genetic variation that alters expression of AIG1 confers greater risk of non-resolving AKI. Funding: NIDDK Support

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.002
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.179
Threshold uncertainty score0.636

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0020.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0020.001
Bibliometrics0.0000.001
Science and technology studies0.0000.001
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.018
GPT teacher head0.247
Teacher spread0.230 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2022
Admission routes1
Has abstractyes

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