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Record W4400895995 · doi:10.1101/2024.07.18.603978

Identification of targetable vulnerabilities of PLK1-overexpressing cancers by synthetic dosage lethality

2024· preprint· en· W4400895995 on OpenAlexafffund
Chelsea E. Cunningham, Frederick S. Vizeacoumar, Yue Zhang, Liliia Kyrylenko, Peng Gao, Vincent Maranda, He Dong, Jared D. W. Price, Ashtalakshmi Ganapathysamy, Rithik Hari, Connor Denomy, Simon Both, Konrad Wagner, Yingwen Wu, Faizaan Khan, Shayla R. Mosley, A. Chen, Tetiana Katrii, Ben G. E. Zoller, Karthic Rajamanickam, Prachi Walke, Lihui Gong, Hardikkumar Patel, Mary Lazell-Wright, Alain Morejon Morales, Kalpana K. Bhanumathy, Hussain Elhasasna, Renuka Dahiya, Omar Abuhussein, Anton E. Dmitriev, Tanya Freywald, Érika Prando Munhoz, Anand Krishnan, Eytan Ruppin, Joo Sang Lee, Katharina Rox, Behzad M. Toosi, Martin Köebel, Mary Kinloch, Laura Hopkins, Cheng‐Han Lee, Raju Datla, Sunil Yadav, Yuliang Wu, Kristi Baker, Martin Empting, Alexandra K. Kiemer, Andrew Freywald, Franco J. Vizeacoumar

Bibliographic record

VenuebioRxiv (Cold Spring Harbor Laboratory) · 2024
Typepreprint
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicDNA Repair Mechanisms
Canadian institutionsUniversity of CalgaryCameco (Canada)Global Institute for Water SecurityAgriculture and Agri-Food CanadaUniversity of SaskatchewanUniversity of AlbertaSaskatchewan Cancer Agency
FundersUniversity of SaskatchewanDeutsche ForschungsgemeinschaftCanadian Institutes of Health ResearchMitacsSaskatchewan Health Research FoundationCancer Research SocietyHuntsman Cancer Institute
KeywordsSynthetic lethalityLethalityIdentification (biology)Cancer researchMedicineChemistryBiologyToxicologyBiochemistryDNA repairDNA

Abstract

fetched live from OpenAlex

Summary Tumor heterogeneity poses a significant challenge in combating treatment resistance. Despite Polo-like kinase 1 (PLK1) being universally overexpressed in cancers and contributing to chromosomal instability (CIN), direct PLK1 inhibition hasn’t yielded clinical progress. To address this, we utilized the synthetic dosage lethality (SDL) approach, targeting PLK1’s genetic interactions for selective killing of overexpressed tumor cells while mitigating heterogeneity-associated challenges. Employing computational methods, we conducted a genome-wide shRNA screen, identifying 105 SDL candidates. Further in vivo CRISPR screening in a breast cancer xenograft model and in vitro CRISPR analysis validated these candidates. Employing Perturb-seq revealed IGF2BP2/IMP2 as a key SDL hit eliminating PLK1-overexpressing cells. Suppression of IGF2BP2, genetically or pharmacologically, downregulated PLK1 and limited tumor growth. Our findings strongly propose targeting PLK1’s genetic interactions as a promising therapeutic approach, holding broad implications across multiple cancers where PLK1 is overexpressed.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.008

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0000.001
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.008
GPT teacher head0.224
Teacher spread0.216 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2024
Admission routes2
Has abstractyes

Explore more

Same venuebioRxiv (Cold Spring Harbor Laboratory)→Same topicDNA Repair Mechanisms→French-language works237,207→