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Record W4403391925 · doi:10.1136/gutjnl-2024-basl.135

P133 The impact of viral genotype in the kinetics hepatitis B surface antigen decline during antiviral therapy, in an East London cohort of chronic hepatitis B patients

2024· article· en· W4403391925 on OpenAlexaboutno aff
Thomas Ngan, Wenhao Li, James J. Cai, Sandhia Naik, Apostolos Koffas, Anna Riddell, Patrick Kennedy, Upkar S. Gill

Bibliographic record

Venuenot available
Typearticle
Languageen
FieldMedicine
TopicHepatitis B Virus Studies
Canadian institutionsnot available
Fundersnot available
KeywordsChronic hepatitisGenotypeCohortMedicineVirologyAntiviral therapyImmunologyAntigenInternal medicineBiologyVirusGeneGenetics

Abstract

fetched live from OpenAlex

The functional cure program for chronic hepatitis B (CHB) is now underway with multiple novel agents entering the clinical trial pipeline. Improved phenotyping of the different CHB disease phases is required for better patient selection for novel therapies. The aim of hepatitis B virus (HBV) functional cure is to achieve sustained loss of hepatitis B surface antigen (HBsAg) following a finite treatment period. Early data from clinical trials demonstrate that lower baseline HBsAg levels may better predict HBsAg loss with novel agents. There are, however limited data with regards to HBV genotype in relation to HBsAg levels. Brouwer et al., reported on the importance of genotype specific HBsAg levels in differentiating disease phases in an American & Canadian cohort (Brower at l., Clin Gastro Heo 2020). Genotype specific HBsAg levels are less well described in a UK (East London) cohort, and if there are specific changes during antiviral therapy. We undertook a retrospective analysis of a diverse cohort of children and adults with CHB infection to determine differences in HBsAg levels between genotypes. A cross-sectional analysis of a sub-cohort was undertaken to determine the dynamic kinetics of HBsAg change during antiviral therapy and the impact of genotype on this. 292 [n=184 male, median age 32 (range 3–69)] consecutive subjects were included in the analysis, of which 183 (n=120 male) subsequently underwent antiviral therapy. Resistance genotyping, viral (HBsAg, HBV DNA, HBeAg status) and biochemical (liver enzymes) parameters were recorded for all subjects. In the overall cohort the mean baseline HBsAg level was 4.61 log10IU/ml. By univariate analysis factors impacting lower HBsAg levels were older age, HBeAg negative disease, lower HBV DNA, antiviral therapy and genotype (all p=<0.05). Prior to commencing therapy no significant difference was noted in HBsAg levels across genotypes, however on antiviral therapy a significant decline in HBsAg was noted in all genotypes. Furthermore we noted the kinetics of on-treatment HBsAg decline was significantly greater in genotype B (p=0.039) and C (p=0.044) vs D (post-hoc ANOVA analysis). Quantitative HBsAg levels are increasingly important in the management of CHB and moreover are used to stratify patients for novel therapies. Our data, from a real world East London cohort, demonstrate variations in HBsAg levels between genotypes on-treatment. Genotype specific HBsAg levels may therefore be important in patient selection for novel therapies or discontuation of treatment trials to achieve functional cure.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.026
Threshold uncertainty score0.052

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.001
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.016
GPT teacher head0.297
Teacher spread0.281 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2024
Admission routes1
Has abstractyes

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