MétaCan
Menu
Back to cohort

Abstract IA002: Leveraging synthetic lethality for the development of novel cancer therapies

2024· article· en· W4405181977 on OpenAlexaboutno aff
Kimberly J. Briggs

Bibliographic record

VenueMolecular Cancer Therapeutics · 2024
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicCancer-related gene regulation
Canadian institutionsnot available
Fundersnot available
KeywordsSynthetic lethalityGenetic screenCancerLethal alleleBiologyRNA interferenceCancer cellGenePopulationMutantCancer researchComputational biologyGeneticsMedicine

Abstract

fetched live from OpenAlex

Abstract The concept of synthetic lethality has long held promise for the next generation of genetically targeted cancer therapies to follow the success of PARP inhibitors in the treatment of BRCA-mutant cancers. The classical definition of synthetic lethality arose in Drosophila with the observation that loss of either of two genes independently had little effect on cell viability but loss of both led to cell death. This definition has now been expanded to capture pairs of genes in which genetic alteration of one gene and pharmacological inhibition of the other leads to death of cancer cells while sparing the normal cells which lack the genetic alteration, leading to a broad therapeutic index. Functional genomics screening (using unbiased RNAi or CRISPR-based technology) has enabled the systematic discovery of novel synthetic lethal targets for drug discovery. The selective dependence of MTAP-deleted cells on PRMT5 is one of the strongest genetic interactions observed in early RNAi screens. Approximately 10-15% of all human cancer is MTAP-deleted, providing a large and diverse patient population. MTA-cooperative PRMT5 inhibitors, which bind preferentially in the presence of MTA, have been developed by scientists at Tango Therapeutics and elsewhere to leverage the synthetic lethal relationship between PRMT5 and MTAP loss. MTA-cooperative PRMT5 inhibitors inhibit PRMT5 in MTAP-deleted cancer cells and spare MTAP-expressing normal cells leading to clinically well-tolerated and efficacious therapies. MTA-cooperative PRMT5 inhibitors have broad therapeutic potential both as single agent and in combination with other therapies. Citation Format: Kimberly Briggs.Leveraging synthetic lethality for the development of novel cancer therapies. [abstract]. In: Proceedings of the AACR Special Conference in Cancer Research: Optimizing Therapeutic Efficacy and Tolerability through Cancer Chemistry; 2024 Dec 9-11; Toronto, Ontario, Canada. Philadelphia (PA): AACR; Mol Cancer Ther 2024;23(12_Suppl):Abstract nr IA002.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Theoretical or conceptual · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: none
Teacher disagreement score0.031
Threshold uncertainty score0.103

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.001
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0010.001
Science and technology studies0.0010.001
Scholarly communication0.0030.002
Open science0.0010.001
Research integrity0.0020.003
Insufficient payload (model declined to judge)0.0310.017

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.045
GPT teacher head0.321
Teacher spread0.276 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designTheoretical or conceptual
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2024
Admission routes1
Has abstractyes

Explore more

Same venueMolecular Cancer TherapeuticsSame topicCancer-related gene regulationFrench-language works237,207