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Record W4406201278 · doi:10.1002/alz.089538

Outdoor air pollution as a risk factor for Alzheimer’s disease: A systematic review

2024· review· en· W4406201278 on OpenAlexaboutno aff
Nehal Hassan, Sarah Wilson, Ríona Mc Ardle, Li Su, Sarah P. Slight

Bibliographic record

VenueAlzheimer s & Dementia · 2024
Typereview
Languageen
FieldEnvironmental Science
TopicAir Quality and Health Impacts
Canadian institutionsnot available
Fundersnot available
KeywordsCINAHLNeuroimagingScopusPopulationMedicineMEDLINEEnvironmental healthDiseaseGerontologyPsychologyPsychiatryPathologyPsychological interventionPolitical science

Abstract

fetched live from OpenAlex

Abstract Background Outdoor air pollution is a global issue which poses a significant health risk. Modern neuroimaging techniques have revealed the detrimental impact of air pollution on brain health, in particular the development and progression of neurodegenerative diseases such as Alzheimer’s disease (AD).(1) We conducted a systematic review to evaluate the effects of long‐term (months to years) exposure to outdoor air pollutants on the development and progression of AD using neuroimaging data. Method This review followed PRISMA guidelines and registered in PROSPERO (CRD42023482979). Four large databases (MEDLINE, Embase, Scopus, and CINAHL) were systematically searched using words relating to “air pollution”, “neuroimaging”, and “Alzheimer’s disease”. The population researched was kept broad to include all ages. There were no geographical limits applied, and so included all countries. Articles were exported to Endnote (Endnote X9.3.3, Clarivate US), where duplicate articles were removed. Remaining articles were uploaded to the Rayyan and screened for eligibility. The Newcastle Ottawa Scale was used to assess the quality of included papers. A narrative synthesis was conducted, which involved grouping papers that focused on the same neuroimaging outcome and comparing and contrasting between studies. Result Our search yielded 397 results, after removing duplicated (n=172), articles were removed at the title (n=192), abstract (n=8), and full text (n=17) stages. Eight articles met our inclusion criteria and focused on changes to white matter (n= 5), cortical thickness (n= 6), and grey matter (n= 2). Specific air pollutants (e.g., PM2.5) were associated with white matter reductions, and PM10 and NO2 with reduced cortical thickness. However, higher exposure to NOx and NO2 was linked to better performance in cognition tests. Exposure to PM2.5 was associated with reduced grey matter, with study participants showing greater cognitive impairment. Air pollution exposure was associated with brain structure changes which are commonly seen in AD‐related pathology. Conclusion Our results highlighted significant associations between specific air pollutant exposure and changes in different brain structures. Future research is needed to further investigate the relationship between air pollution exposure and cognitive decline. References: Block ML, Calderón‐Garcidueñas L. Air pollution: mechanisms of neuroinflammation and CNS disease. Trends Neurosci. 2009;32(9):506‐16.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.008
metaresearch head score (Gemma)0.034
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Systematic review · Consensus signal: Systematic review
GenreCandidate signal: Review · Consensus signal: Review
Teacher disagreement score0.009
Threshold uncertainty score0.043

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0080.034
Meta-epidemiology (narrow)0.0010.001
Meta-epidemiology (broad)0.0080.006
Bibliometrics0.0090.011
Science and technology studies0.0010.001
Scholarly communication0.0030.002
Open science0.0020.001
Research integrity0.0020.001
Insufficient payload (model declined to judge)0.0050.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.102
GPT teacher head0.387
Teacher spread0.285 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designSystematic review
Domainnot available
GenreReview

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations2
Published2024
Admission routes1
Has abstractyes

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