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Record W4406218433 · doi:10.1002/alz.087481

Multivariate genomic analyses of frailty reveal the unique importance of cognitive and multimorbidity pathways for aging‐related health outcomes

2024· article· en· W4406218433 on OpenAlexaff
Isabelle F. Foote, Jonny P Flint, John D. Fisk, Tobias K. Karakach, Andrew D. Rutenberg, Nicholas G. Martin, Michelle K. Lupton, Michelle Luciano, Kenneth Rockwood, Andrew D. Grotzinger

Bibliographic record

VenueAlzheimer s & Dementia · 2024
Typearticle
Languageen
FieldMedicine
TopicFrailty in Older Adults
Canadian institutionsNova Scotia Health AuthorityDalhousie University
Fundersnot available
KeywordsMultimorbidityMultivariate statisticsCognitionGerontologyCognitive agingPsychologyMultivariate analysisData scienceComorbidityMedicinePsychiatryComputer scienceInternal medicine

Abstract

fetched live from OpenAlex

Abstract Background Frailty is a complex clinical state that is associated with poorer health outcomes and increased dementia risk in older adults. It is routinely measured using the Frailty Index, which is a proportional score based on the number of ‘deficits’ that an individual has. Whilst such measures are useful for risk assessment, the aggregation of highly heterogeneous deficit profiles in genetic studies may obscure important insights into the underlying biology of frailty. Method We used Genomic Structural Equation Modelling to model shared genetic signal between 30 deficits from the Frailty Index using summary data from genome‐wide association studies (GWAS). This involved conducting a multivariate GWAS to identify genomic risk loci associated with each of the latent frailty factors in our model. We used polygenic risk scores (PRS) to test the predictive accuracy of the latent factors for detecting frailty status in 3 external cohort studies. Finally, we tested the genetic correlation between our latent frailty factors and >50 aging‐related outcomes, including dementia. Result The genetic architecture of the frailty deficits was best captured by a general factor influencing all 30 deficits, plus 6 additional latent factors representing genetic overlap between distinct subsets of deficits that were uncorrelated with the general factor. These 6 factors can be broadly described as reflecting pathways linked to social isolation, unhealthy lifestyle, multimorbidity, metabolic/respiratory status, cognition, and disability. GWAS conducted to measure the effects of individual genetic variants on these frailty factors identified 408 genomic risk loci. Our PRS analyses demonstrated consistent evidence that shared genetic pathways between frailty deficits linked to multimorbidity and cognition, but unique of the general frailty factor, are particularly important for predicting frailty status. Genetic correlation analyses further highlighted a significant genetic association between frailty pathways linked to cognition and Alzheimer’s disease (rg = 0.32, SE = 0.07, p = 5.58×10−06). Conclusion Our findings provide novel insights into the characterization of general and specific pathways within the frailty state at the genetic level. We demonstrate how refining our knowledge of frailty, particularly in relation to multimorbidity and cognitive impairment, may help to stratify frail individuals at increased risk of developing dementia or other adverse health outcomes.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.002
metaresearch head score (Gemma)0.007
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.013
Threshold uncertainty score0.026

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0020.007
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0000.002
Bibliometrics0.0010.001
Science and technology studies0.0010.001
Scholarly communication0.0010.000
Open science0.0010.001
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0030.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.111
GPT teacher head0.389
Teacher spread0.279 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2024
Admission routes1
Has abstractyes

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