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Record W4406687355 · doi:10.1093/ecco-jcc/jjae190.0172

DOP133 Spatiotemporal analysis of Crohn’s disease reveals PECAM2 signaling at the basis of inflammation-to-fibrosis transition

2025· article· en· W4406687355 on OpenAlexaff
Federica Ungaro, Luca Massimino, Tommaso Lorenzo Parigi, Mattéo Riva, S Nicolò, Salvatore Spanò, S Mino, Mattia Bugatti, Alice Frontali, F Scarfò, Andrea Vignali, Andrea Municchi, V. Villanacci, Luca Albarello, Mirco Ponzoni, Virginia Solitano, Vipul Jairath, Laurent Peyrin‐Biroulet, Pierpaolo Sileri, Silvio Danese

Bibliographic record

VenueJournal of Crohn s and Colitis · 2025
Typearticle
Languageen
FieldMedicine
TopicCell Adhesion Molecules Research
Canadian institutionsWestern University
Fundersnot available
KeywordsMedicineCrohn's diseaseInflammationFibrosisDiseaseInflammatory bowel diseaseTransition (genetics)ImmunologyInternal medicine

Abstract

fetched live from OpenAlex

Abstract Background Crohn’s disease is a chronic inflammatory disease of the bowel often complicated by fibrotic strictures(1,2,3). Medical treatment is lacking, and surgery is commonly required(3). The mechanisms underlying the progression from chronic inflammation to fibrosis are not yet defined. We aim to unravel Crohn’s disease pathogenesis using cutting-edge computational and bioinformatics tools (Figure 1). Methods Spatial transcriptomics was performed on 13 surgical specimens of inflamed and fibrotic Crohn’s disease and healthy controls. Spatial transcriptomics results were integrated with single-cell data to track the cellular and molecular evolution from healthy intestine to fibrosis by employing CellChat(4) and pseudotime analysis. Computational data were confirmed by immunostaining of tissues from an independent cohort of Crohn’s patients. Results We demonstrated that intestinal cytoarchitecture was rearranged while chronic inflammation progressed. Crohn’s disease-associated fibrosis evolved within the mesenchymal compartment, driven by PECAM2 signaling through PECAM1-CD38 interaction. Notably, CD38 positivity was found in the mesenchymal compartment in an independent cohort of Crohn’s disease patients. In parallel, ApoA signaling, particularly APOA1-ABCA interaction, emerged as relevant for maintaining epithelial and stromal homeostasis, while its downregulation was associated with fibrosis development (Figure 2). Conclusion Our results provide insights into CD38-driven fibrosis and suggest that PECAM2 signaling blockade could reduce the development of strictures in patients with Crohn’s disease, potentially offering a new treatment target. References 1.Torres J, Mehandru S, Colombel J-F, et al. Crohn’s disease. Lancet 2017;389:1741–1755. 2.Lichtenstein GR, Loftus EV, Isaacs KL, et al. ACG clinical guideline: management of crohn’s disease in adults. Am. J. Gastroenterol. 2018;113:481–517. 3.Rieder F, Kessler S, Sans M, et al. Animal models of intestinal fibrosis: new tools for the understanding of pathogenesis and therapy of human disease. Am. J. Physiol. Gastrointest. Liver Physiol. 2012;303:G786-801. 4.Jin S, Plikus MV, Nie Q. CellChat for systematic analysis of cell-cell communication from single-cell and spatially resolved transcriptomics. BioRxiv 2023.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.003

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.014
GPT teacher head0.297
Teacher spread0.282 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2025
Admission routes1
Has abstractyes

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