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Record W4407285718 · doi:10.1093/jcag/gwae059.152

A152 INTESTINAL INFLAMMATION DISRUPTS MICROBIAL ANTIGEN METABOLISM FACILITATING FOOD SENSITIZATION

2025· article· en· W4407285718 on OpenAlexaff
B Barbosa da Luz, L Rondeau, Pranshu Muppidi, Rammy Dang, Alberto Caminero

Bibliographic record

VenueJournal of the Canadian Association of Gastroenterology · 2025
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicDigestive system and related health
Canadian institutionsMcMaster University
Fundersnot available
KeywordsSensitizationInflammationAntigenMetabolismImmunologyMicrobiologyBiologyBiochemistry

Abstract

fetched live from OpenAlex

Abstract Background Patients with inflammatory bowel disease (IBD) report diverse food-related adverse reactions. Dairy is the most self-reported food intolerance in IBD. Emerging evidence suggests that altered diet-microbiota interactions can contribute to the development of IBD and adverse reaction to foods. We hypothesize that microbial alterations in IBD patients facilitate adverse reactions to foods. Aims To investigate the role of intestinal inflammation on microbial metabolism and food sensitization. Methods To investigate the effects of inflammation on microbial metabolism of food, eight-week-old C57BL6 mice were exposed to different concentrations (1.5 – 3.5%) of dextran sodium sulfate (DSS) for 5 days, then gavaged with dairy proteins. To study the role of inflammation in food sensitization, C57BL/6 mice were exposed to 3% DSS for 5 days, then sensitized to dairy proteins (6 mg casein/whey protein) using 4 oral administrations of cholera toxin (20 ug/mouse). After 1 week of recovery, sensitized mice were exposed to either a dairy-free or dairy containing diet for 7 days. Finally, the effects of inflammatory-mediated microbial alterations on food sensitivities were studied in gnotobiotic mice. Germ-free C57BL6 were colonized with DSS-inflamed microbiota or healthy SPF microbiota. After 3 weeks of colonization, the mice were sensitized to dairy proteins as described above, exposed to dairy and one cycle of DSS. Intestinal microbiota composition (16S sequencing) and function (digestion assays), and markers of intestinal inflammation (disease activity index, histological damage, cell infiltration and pro-inflammatory genes expression) were analyzed. Results DSS led to pronounced changes in the intestinal microbiota, impaired microbial capacity to degrade dairy proteins and increased systemic access of antigens. Inflammation also facilitated food sensitization characterized by immune cell infiltration (polymorphonuclear cells and CD3+) and mast cell proliferation in the colonic mucosa, and generation of systemic IgE and IgG levels. Dairy increased genes associated to immune cell recruitment and apoptosis in sensitized mice, and increased of apoptosis in the colon was confirmed by staining (TUNEL). Finally, mice colonized with microbiota from DSS-inflamed mice showed a worsening in inflammatory parameters during the dairy-diet intervention after sensitization. These results suggest that microbial alterations after inflammation facilitate the onset of food sensitivities and exacerbate food-mediated immune reactions in the colon. Conclusions Intestinal inflammation facilitates sensitization to foods by altering microbial antigen metabolism in the colon. Funding Agencies CCC

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.001
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.113
Threshold uncertainty score0.923

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0010.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.004
GPT teacher head0.221
Teacher spread0.217 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2025
Admission routes1
Has abstractyes

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